Literature DB >> 2967683

Lack of inhibition of glycolytic enzymes by the neurotoxic organophosphorus compounds mipafox and methamidofos.

A F Hernández1, A Pla, E Villanueva.   

Abstract

Preincubation with Mipafox and Methamidofos as well as Paraoxon (used as control) did not cause inhibition of hexokinase, phosphofructokinase, glyceraldehyde-3-phosphate dehydrogenase and lactate dehydrogenase. This is in contrast with the inhibition of glycolysis by other neurotoxic compounds (hexacarbons, acrylamide, carbon disulfide).

Entities:  

Mesh:

Substances:

Year:  1988        PMID: 2967683     DOI: 10.1007/bf00364859

Source DB:  PubMed          Journal:  Arch Toxicol        ISSN: 0340-5761            Impact factor:   5.153


  3 in total

Review 1.  The delayed neuropathy caused by some organophosphorus esters: mechanism and challenge.

Authors:  M K Johnson
Journal:  CRC Crit Rev Toxicol       Date:  1975-06

2.  Postmortem activity of the key enzymes of glycolysis. In rat brain regions in relation to time after death.

Authors:  A Pla; L Lemus; A Valenzuela; E Villanueva
Journal:  Z Rechtsmed       Date:  1986

3.  Studies on the biochemical basis of distal axonopathies--I. Inhibition of glycolysis by neurotoxic hexacarbon compounds.

Authors:  M I Sabri; C L Moore; P S Spencer
Journal:  J Neurochem       Date:  1979-03       Impact factor: 5.372

  3 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.