| Literature DB >> 29669588 |
Man-Qing Cao1,2, A-Bin You3, Xiao-Dong Zhu1, Wei Zhang2, Yuan-Yuan Zhang1, Shi-Zhe Zhang1, Kei-Wei Zhang4, Hao Cai1, Wen-Kai Shi1, Xiao-Long Li1, Kang-Shuai Li1, Dong-Mei Gao1, De-Ning Ma5, Bo-Gen Ye6, Cheng-Hao Wang7, Cheng-Dong Qin8, Hui-Chan Sun9, Ti Zhang10, Zhao-You Tang11.
Abstract
The original article [1] contains an error in Fig. 5a whereby the Western blot bands representing CyclinD1 have mistakenly been duplicated over the Western blot bands intended to represent SGK.Entities:
Year: 2018 PMID: 29669588 PMCID: PMC5907218 DOI: 10.1186/s13045-018-0599-z
Source DB: PubMed Journal: J Hematol Oncol ISSN: 1756-8722 Impact factor: 17.388
Fig. 5FOXO3a exerts an important mediator in miR-182-5p induced Wnt signaling activation. a Western blot analysis of Wnt signaling pathway related proteins following miR-182-5p overexpression and knockdown. b IHC staining of FOXO3a and β-catenin in orthotopic tumor tissues of negative control and overexpression of miR-182-5p. c Western blot analysis of c-Myc in miR-NC cells treated with different concentration of XAV939 (0, 1, 5, 10, 100, and 200 μM). d Transwell assay of HCC cells that transfected with miR-NC, overexpression of miR-182-5p and miR-182-5p overexpression cells that further treated with 10 μM XAV939. e Western blot analysis of Wnt signaling pathway-related proteins in miR-NC cells, overexpression of miR-182-5p cells and miR-182-5p overexpression cells further overexpress FOXO3a