Literature DB >> 29655763

Sleeping oxygen saturation, rapid eye movement sleep, and the adaptation of postprandial metabolic function in insulin sensitive and resistant individuals without diabetes.

Karin A Garcia1, William K Wohlgemuth2, Ele Ferrannini3, Andrea Mari4, Alex Gonzalez5, Armando J Mendez6, Roberto Bizzotto4, Jay S Skyler7, Neil Schneiderman8, Barry E Hurwitz9.   

Abstract

AIMS: Sleeping oxygen saturation (SaO2) and sleep stage duration have been linked with prediabetic alterations but the pathogenic pathways are not well understood. This study of insulin sensitive and resistant adults examined the effect on postprandial metabolic regulation of repeated mixed-meal challenges of different carbohydrate loading. The aim was to examine whether the relationship between lower sleeping oxygen saturation (SaO2) and poorer fasting and postprandial metabolic function may be linked with reduced slow wave sleep (SWS) and rapid eye movement (REM) duration, independent of age, sex and total adiposity.
METHODS: The 24 men and women, aged 25-54 years, had no diabetes or other diagnosed conditions, were evaluated with polysomnography to derive indices of SaO2 and sleep architecture. In addition, an OGTT and two 14-h serial mixed-meal tests were administered over 3 successive in-patient days. The carbohydrate content of the mixed-meals was manipulated to compare a standard-load day with a double-load day (300 vs. 600 kcal/meal). Quantitative modeling was applied to derive β-cell glucose sensitivity (β-GS), early insulin secretion rate sensitivity (ESRS), and total postprandial insulinemia (AUCINS).
RESULTS: Analyses showed that, for the 14-h tests, the SaO2 relationship with metabolic outcomes was associated significantly with percent time spent in REM but not SWS, independent of age, sex and total adiposity. Specifically, indirect pathways indicated that lower SaO2 was related to shorter REM duration, and shorter REM was respectively associated with higher β-GS, ESRS, and AUCINS for the 300- and 600-load days (300 kcal/meal: β = -8.68, p < .03, β = -8.54, p < .002, and β = -10.06, p < .008; 600 kcal/meal: β = -11.45, p < .003, β = -11.44, p < .001, and β = -11.00, p < .03).
CONCLUSION: Sleeping oxygen desaturation and diminished REM duration are associated with a metabolic pattern that reflects a compensatory adaptation of postprandial insulin metabolism accompanying preclinical diabetic risk.
Copyright © 2018 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Glycemia; Insulin resistance; Insulin secretion; Postprandial; REM; Sleeping oxygen saturation

Mesh:

Substances:

Year:  2018        PMID: 29655763      PMCID: PMC5975635          DOI: 10.1016/j.physbeh.2018.04.009

Source DB:  PubMed          Journal:  Physiol Behav        ISSN: 0031-9384


  51 in total

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Authors:  Salha Fendri; Dominique Rose; Sonia Myambu; Sandrine Jeanne; Jean-Daniel Lalau
Journal:  Diabetes Res Clin Pract       Date:  2011-01       Impact factor: 5.602

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Journal:  Clin Sci (Lond)       Date:  2017-12-04       Impact factor: 6.124

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Journal:  Diabetes       Date:  1979-12       Impact factor: 9.461

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Authors:  Riva Tauman; Louise M O'Brien; David Gozal
Journal:  Sleep Breath       Date:  2007-06       Impact factor: 2.655

9.  Sleep architecture and glucose and insulin homeostasis in obese adolescents.

Authors:  Dorit Koren; Lorraine E Levitt Katz; Preneet C Brar; Paul R Gallagher; Robert I Berkowitz; Lee J Brooks
Journal:  Diabetes Care       Date:  2011-09-20       Impact factor: 17.152

10.  Adaptation of β-Cell and Endothelial Function to Carbohydrate Loading: Influence of Insulin Resistance.

Authors:  Barry E Hurwitz; Neil Schneiderman; Jennifer B Marks; Armando J Mendez; Alex Gonzalez; Maria M Llabre; Steven R Smith; Roberto Bizzotto; Eleonora Santini; Maria Laura Manca; Jay S Skyler; Andrea Mari; Ele Ferrannini
Journal:  Diabetes       Date:  2015-03-09       Impact factor: 9.461

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