Literature DB >> 29655081

PI3K-AKT-GSK3β-CREB signaling pathway regulates anxiety-like behavior in rats following alcohol withdrawal.

Xiaomeng Qiao1, Haiyun Gai2, Rui Su1, Cuola Deji1, Jingjing Cui1, Jianghua Lai3, Yongsheng Zhu4.   

Abstract

BACKGROUND: Alcohol abuse and anxiety disorders often occur concurrently, but their underlying cellular mechanisms remain unclear. Neuroadaptation within the medial prefrontal cortex (mPFC) have been implicated in the molecular mechanisms underlying alcohol drinking behavior and withdrawal.
METHODS: A chronic alcohol exposure rat model (35 consecutive days of 10% alcohol intake and 48 h of withdrawal) was established, then, wortmannin (0.5 µg/side) was injected bilaterally into the mPFC. The elevated plus maze (EPM) and open field test (OFT) were used to assess anxiety-like behavior. Western blot assays were used to assess protein levels.
RESULTS: We found that anxiety-like behavior peaked approximately 6 h after alcohol withdrawal. However, wortmannin greatly decreased alcohol intake and attenuated anxiety-like behavior in the alcohol exposure rats. Moreover, the PI3K-AKT-GSK3β signaling pathway was activated after alcohol withdrawal, and phosphorylation of the downstream cAMP response element-binding protein (CREB) was increased. Wortmannin uniformly reversed PI3K-AKT-GSK3β-CREB pathway phosphorylation. LIMITATIONS: The downstream GSK3β activity was not intervened and a single dose level of wortmannin was used.
CONCLUSION: Our results suggest that activating the PI3K-AKT-GSK3β-CREB pathway in the mPFC is an important contributor to the molecular mechanisms underlying alcohol withdrawal. PI3K signaling pathway inhibitors are thus potential candidates for treating alcohol abuse.
Copyright © 2018 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Alcohol withdrawal; Anxiety-like behavior; PI3K-AKT-GSK3β-CREB signaling pathway; The medial prefrontal cortex; Wortmannin

Mesh:

Substances:

Year:  2018        PMID: 29655081     DOI: 10.1016/j.jad.2018.04.039

Source DB:  PubMed          Journal:  J Affect Disord        ISSN: 0165-0327            Impact factor:   4.839


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