Literature DB >> 29625343

Rapid exhaustion of auditory neural conduction in a prototypical mitochondrial disease, Friedreich ataxia.

Fabrice Giraudet1, Perrine Charles2, Thierry Mom1, Odile Boespflug-Tanguy3, Alexandra Dürr4, Paul Deltenre5, Paul Avan6.   

Abstract

OBJECTIVES: In patients with Friedreich ataxia (FRDA), mitochondrial failure leads to impaired cellular energetics. Since many FRDA patients have impaired hearing in noise, we investigated the objective consequences on standard auditory brainstem-evoked responses (ABRs).
METHODS: In 37 FRDA patients, among whom 34 with abnormal standard ABRs, hearing sensitivity, speech-in-noise intelligibility and otoacoustic emissions were controlled. ABR recordings were split into four consecutive segments of the total time frame used for data collection, thus allowing the dynamics of ABR averaging to be observed.
RESULTS: Most ears showed features of an auditory neuropathy spectrum disorder with flattened ABRs and impaired speech-in-noise intelligibility contrasting with near-normal hearing sensitivity and normal preneural responses. Yet split-ABRs revealed short-lived wave patterns in 26 out of 68 ears with flattened standard ABRs (38%). While averaging went on, the pattern of waves shifted so that interwave latencies increased by 35% on average.
CONCLUSIONS: In FRDA, the assumption of stationarity used for extracting standard ABRs is invalid. The preservation of early split-ABRs indicates no short-term dyssynchrony of action potentials. A large decrease in conduction velocity along auditory neurons occurs within seconds, attributed to fast energetic failure. SIGNIFICANCE: This model of metabolic sensory neuropathy warns against exposure of metabolically-impaired patients to sustained auditory stimulation.
Copyright © 2018 International Federation of Clinical Neurophysiology. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Auditory brainstem; Conduction velocity; Friedreich ataxia; Neurodegeneration

Mesh:

Year:  2018        PMID: 29625343     DOI: 10.1016/j.clinph.2018.03.005

Source DB:  PubMed          Journal:  Clin Neurophysiol        ISSN: 1388-2457            Impact factor:   3.708


  4 in total

1.  In vivo survival and differentiation of Friedreich ataxia iPSC-derived sensory neurons transplanted in the adult dorsal root ganglia.

Authors:  Serena Viventi; Stefano Frausin; Sara E Howden; Shiang Y Lim; Rocio K Finol-Urdaneta; Jeffrey R McArthur; Kwaku Dad Abu-Bonsrah; Wayne Ng; Jason Ivanusic; Lachlan Thompson; Mirella Dottori
Journal:  Stem Cells Transl Med       Date:  2021-03-18       Impact factor: 6.940

2.  Oral mobility reflects rate of progression in advanced Friedreich's ataxia.

Authors:  Stéphanie Borel; Peggy Gatignol; Mustapha Smail; Marie-Lorraine Monin; Claire Ewenczyk; Didier Bouccara; Alexandra Durr
Journal:  Ann Clin Transl Neurol       Date:  2019-08-25       Impact factor: 4.511

3.  Vestibular impact of Friedreich ataxia in early onset patients.

Authors:  A Maudoux; N Teissier; M Francois; Th Van Den Abbeele; C Alberti; I Husson; S R Wiener-Vacher
Journal:  Cerebellum Ataxias       Date:  2020-05-28

4.  Auditory Phenotypic Variability in Friedreich's Ataxia Patients.

Authors:  Nehzat Koohi; Gilbert Thomas-Black; Paola Giunti; Doris-Eva Bamiou
Journal:  Cerebellum       Date:  2021-02-18       Impact factor: 3.847

  4 in total

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