Literature DB >> 29611167

Proteomics study of serum exosomes in Kawasaki disease patients with coronary artery aneurysms.

Xiao-Fei Xie1, Hong-Juan Chu2, Yu-Fen Xu1, Liang Hua1, Zhou-Ping Wang1, Ping Huang1, Hong-Ling Jia3, Li Zhang4.   

Abstract

BACKGROUND: To study the protein profile of the serum exosomes of patients with coronary artery aneurysms (CAA) caused by Kawasaki disease (KD).
METHODS: Two-dimensional electrophoresis (2-DE) was used to identify proteins from the exosomes of serum obtained from children with CAA caused by KD, as well as healthy controls. Differentially expressed proteins were identified using matrix-assisted laser desorption/ionization time-of-flight/timeof-flight mass spectrometry (MALDI-TOF/TOF MS) analysis.
RESULTS: Thirty two differentially expressed proteins were identified (18 up-regulated and 14 downregulated) from serum exosomes of children with CAA and were compared to healthy controls. The expression levels of 4 proteins (TN, RBP4, LRG1, and APOA4) were validated using Western blotting. Classification analysis and protein-protein network analysis showed that they are associated with multiple functional groups, including host immune response, inflammation, apoptotic process, developmental process, and biological adhesion process.
CONCLUSIONS: These findings establish a comprehensive proteomic profile of serum exosomes from children with CAA caused by KD, and provide additional insights into the mechanisms of CAA caused by KD.

Entities:  

Keywords:  Kawasaki disease; coronary aneurysm; exosome; proteomics

Mesh:

Substances:

Year:  2018        PMID: 29611167      PMCID: PMC8084394          DOI: 10.5603/CJ.a2018.0032

Source DB:  PubMed          Journal:  Cardiol J        ISSN: 1898-018X            Impact factor:   2.737


  28 in total

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Journal:  Molecules       Date:  2019-09-27       Impact factor: 4.411

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6.  An Immunological Axis Involving Interleukin 1β and Leucine-Rich-α2-Glycoprotein Reflects Therapeutic Response of Children with Kawasaki Disease: Implications from the KAWAKINRA Trial.

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7.  Leucine-Rich α-2-Glycoprotein 1 Suppresses Endothelial Cell Activation Through ADAM10-Mediated Shedding of TNF-α Receptor.

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Review 8.  LRG1: an emerging player in disease pathogenesis.

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  8 in total

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