Literature DB >> 29606310

Biophysical Properties of Extracellular Matrix: Linking Obesity and Cancer.

Joseph E Druso1, Claudia Fischbach2.   

Abstract

Obesity has been associated with increased severity of diagnoses for several types of cancer, and recent evidence suggests that the mechanism by which obese tissues contribute to cancer progression involves the extracellular matrix (ECM). Understanding the physicochemical differences between lean and obese ECM, and how cancer cells respond to these differences, promises therapeutic insight.
Copyright © 2018 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  cancer; extracellular matrix; obesity

Mesh：

Substances：

Year:  2018        PMID： 29606310      PMCID： PMC5884448          DOI： 10.1016/j.trecan.2018.02.001

Source DB:  PubMed          Journal:  Trends Cancer        ISSN： 2405-8025


  15 in total

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3.  Obesity-Induced Inflammation and Desmoplasia Promote Pancreatic Cancer Progression and Resistance to Chemotherapy.

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Review 5.  Obesity and Cancer Mechanisms: Cancer Metabolism.

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6.  Contact guidance mediated three-dimensional cell migration is regulated by Rho/ROCK-dependent matrix reorganization.

Authors:  Paolo P Provenzano; David R Inman; Kevin W Eliceiri; Steven M Trier; Patricia J Keely
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Review 7.  Hyaluronan, fluid pressure, and stromal resistance in pancreas cancer.

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Review 10.  Force-dependent cell signaling in stem cell differentiation.

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  13 in total

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Review 5.  Weighing the Risk: effects of Obesity on the Mammary Gland and Breast Cancer Risk.

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Review 6.  Tumour Initiation: a Discussion on Evidence for a "Load-Trigger" Mechanism.

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7.  Compression-induced dedifferentiation of adipocytes promotes tumor progression.

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9.  High-fat diet-fed ovariectomized mice are susceptible to accelerated subcutaneous tumor growth potentially through adipose tissue inflammation, local insulin-like growth factor release, and tumor associated macrophages.

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10.  Context-dependent selection as the keystone in the somatic evolution of cancer.

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