Literature DB >> 29601269

Molecular Minimal Residual Disease in Acute Myeloid Leukemia.

Mojca Jongen-Lavrencic1, Tim Grob1, Diana Hanekamp1, François G Kavelaars1, Adil Al Hinai1, Annelieke Zeilemaker1, Claudia A J Erpelinck-Verschueren1, Patrycja L Gradowska1, Rosa Meijer1, Jacqueline Cloos1, Bart J Biemond1, Carlos Graux1, Marinus van Marwijk Kooy1, Markus G Manz1, Thomas Pabst1, Jakob R Passweg1, Violaine Havelange1, Gert J Ossenkoppele1, Mathijs A Sanders1, Gerrit J Schuurhuis1, Bob Löwenberg1, Peter J M Valk1.   

Abstract

BACKGROUND: Patients with acute myeloid leukemia (AML) often reach complete remission, but relapse rates remain high. Next-generation sequencing enables the detection of molecular minimal residual disease in virtually every patient, but its clinical value for the prediction of relapse has yet to be established.
METHODS: We conducted a study involving patients 18 to 65 years of age who had newly diagnosed AML. Targeted next-generation sequencing was carried out at diagnosis and after induction therapy (during complete remission). End points were 4-year rates of relapse, relapse-free survival, and overall survival.
RESULTS: At least one mutation was detected in 430 out of 482 patients (89.2%). Mutations persisted in 51.4% of those patients during complete remission and were present at various allele frequencies (range, 0.02 to 47%). The detection of persistent DTA mutations (i.e., mutations in DNMT3A, TET2, and ASXL1), which are often present in persons with age-related clonal hematopoiesis, was not correlated with an increased relapse rate. After the exclusion of persistent DTA mutations, the detection of molecular minimal residual disease was associated with a significantly higher relapse rate than no detection (55.4% vs. 31.9%; hazard ratio, 2.14; P<0.001), as well as with lower rates of relapse-free survival (36.6% vs. 58.1%; hazard ratio for relapse or death, 1.92; P<0.001) and overall survival (41.9% vs. 66.1%; hazard ratio for death, 2.06; P<0.001). Multivariate analysis confirmed that the persistence of non-DTA mutations during complete remission conferred significant independent prognostic value with respect to the rates of relapse (hazard ratio, 1.89; P<0.001), relapse-free survival (hazard ratio for relapse or death, 1.64; P=0.001), and overall survival (hazard ratio for death, 1.64; P=0.003). A comparison of sequencing with flow cytometry for the detection of residual disease showed that sequencing had significant additive prognostic value.
CONCLUSIONS: Among patients with AML, the detection of molecular minimal residual disease during complete remission had significant independent prognostic value with respect to relapse and survival rates, but the detection of persistent mutations that are associated with clonal hematopoiesis did not have such prognostic value within a 4-year time frame. (Funded by the Queen Wilhelmina Fund Foundation of the Dutch Cancer Society and others.).

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Year:  2018        PMID: 29601269     DOI: 10.1056/NEJMoa1716863

Source DB:  PubMed          Journal:  N Engl J Med        ISSN: 0028-4793            Impact factor:   91.245


  189 in total

1.  Targeted next-generation sequencing of circulating cell-free DNA vs bone marrow in patients with acute myeloid leukemia.

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Review 2.  Clinical consequences of clonal hematopoiesis of indeterminate potential.

Authors:  David P Steensma
Journal:  Hematology Am Soc Hematol Educ Program       Date:  2018-11-30

Review 3.  When to obtain genomic data in acute myeloid leukemia (AML) and which mutations matter.

Authors:  Gregory W Roloff; Elizabeth A Griffiths
Journal:  Hematology Am Soc Hematol Educ Program       Date:  2018-11-30

Review 4.  Energy metabolism and drug response in myeloid leukaemic stem cells.

Authors:  Alfonso E Bencomo-Alvarez; Andres J Rubio; Mayra A Gonzalez; Anna M Eiring
Journal:  Br J Haematol       Date:  2019-06-24       Impact factor: 6.998

5.  Assessment of Molecular Relapse Detection in Early-Stage Breast Cancer.

Authors:  Isaac Garcia-Murillas; Neha Chopra; Iñaki Comino-Méndez; Matthew Beaney; Holly Tovey; Rosalind J Cutts; Claire Swift; Divya Kriplani; Maria Afentakis; Sarah Hrebien; Giselle Walsh-Crestani; Peter Barry; Stephen R D Johnston; Alistair Ring; Judith Bliss; Simon Russell; Abigail Evans; Anthony Skene; Duncan Wheatley; Mitch Dowsett; Ian E Smith; Nicholas C Turner
Journal:  JAMA Oncol       Date:  2019-10-01       Impact factor: 31.777

6.  Impact of preleukemic mutations and their persistence on hematologic recovery after induction chemotherapy for AML.

Authors:  Tracy Murphy; Jinfeng Zou; Georgina S Daher-Reyes; Andrea Arruda; Vikas Gupta; Caroline J McNamara; Mark D Minden; Aaron D Schimmer; Hassan Sibai; Karen W L Yee; Mariam Korulla; Tracy Stockley; Suzanne Kamel-Reid; Dawn Maze; Anne Tierens; Scott V Bratman; Andre C Schuh; Steven M Chan
Journal:  Blood Adv       Date:  2019-08-13

7.  Response assessment in acute myeloid leukemia by flow cytometry supersedes cytomorphology at time of aplasia, amends cases without molecular residual disease marker and serves as an independent prognostic marker at time of aplasia and post-induction.

Authors:  Thomas Köhnke; Veit Bücklein; Sandra Rechkemmer; Stephanie Schneider; Maja Rothenberg-Thurley; Klaus H Metzeler; Maria-Cristina Sauerland; Wolfgang Hiddemann; Karsten Spiekermann; Marion Subklewe
Journal:  Haematologica       Date:  2019-04-04       Impact factor: 9.941

8.  FLT3 Inhibitor Maintenance After Allogeneic Transplantation: Is a Placebo-Controlled, Randomized Trial Ethical?

Authors:  Mark J Levis; Yi-Bin Chen; Mehdi Hamadani; Mary M Horowitz; Richard J Jones
Journal:  J Clin Oncol       Date:  2019-04-29       Impact factor: 44.544

Review 9.  Clinical consequences of clonal hematopoiesis of indeterminate potential.

Authors:  David P Steensma
Journal:  Blood Adv       Date:  2018-11-27

10.  NPM1 mutated AML can relapse with wild-type NPM1: persistent clonal hematopoiesis can drive relapse.

Authors:  Alexander Höllein; Manja Meggendorfer; Frank Dicker; Sabine Jeromin; Niroshan Nadarajah; Wolfgang Kern; Claudia Haferlach; Torsten Haferlach
Journal:  Blood Adv       Date:  2018-11-27
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