Literature DB >> 29514830

Progenitor Cells and Clinical Outcomes in Patients With Acute Coronary Syndromes.

Ayman Samman Tahhan1,2, Muhammad Hammadah1,2, Mohamad Raad1,2, Zakaria Almuwaqqat1,2, Ayman Alkhoder1,2, Pratik B Sandesara1,2, Heval Mohamed-Kelli1,2, Salim S Hayek1,2, Jeong Hwan Kim1,2, Wesley T O'Neal1,2, Matthew L Topel1,2, Aubrey J Grant1,2, Nabil Sabbak1,2, Robert E Heinl1,2, Mohamad Mazen Gafeer1,2, Malik Obideen1,2, Belal Kaseer1,2, Nasser Abdelhadi1,2, Yi-An Ko1,2,3, Chang Liu3, Iraj Hesaroieh1,2, Ernestine A Mahar3, Viola Vaccarino1,2, Edmund K Waller4, Arshed A Quyyumi5,2.   

Abstract

RATIONALE: Circulating progenitor cells (CPCs) mobilize in response to ischemic injury, but their predictive value remains unknown in acute coronary syndrome (ACS).
OBJECTIVE: We aimed to investigate the number of CPCs in ACS compared with those with stable coronary artery disease (CAD), relationship between bone marrow PCs and CPCs, and whether CPC counts predict mortality in patients with ACS. METHODS AND
RESULTS: In 2028 patients, 346 had unstable angina, 183 had an acute myocardial infarction (AMI), and the remaining 1499 patients had stable CAD. Patients with ACS were followed for the primary end point of all-cause death. CPCs were enumerated by flow cytometry as mononuclear cells expressing a combination of CD34+, CD133+, vascular endothelial growth factor receptor 2+, or chemokine (C-X-C motif) receptor 4+. CPC counts were higher in subjects with AMI compared those with stable CAD even after adjustment for age, sex, race, body mass index, renal function, hypertension, diabetes mellitus, hyperlipidemia, and smoking; CD34+, CD34+/CD133+, CD34+/CXCR4+, and CD34+/VEGFR2+ CPC counts were 19%, 25%, 28%, and 142% higher in those with AMI, respectively, compared with stable CAD. There were strong correlations between the concentrations of CPCs and the PC counts in bone marrow aspirates in 20 patients with AMI. During a 2 (interquartile range, 1.31-2.86)-year follow-up period of 529 patients with ACS, 12.4% died. In Cox regression models adjusted for age, sex, body mass index, heart failure history, estimated glomerular filtration rate, and AMI, subjects with low CD34+ cell counts had a 2.46-fold (95% confidence interval, 1.18-5.13) increase in all-cause mortality, P=0.01. CD34+/CD133+ and CD34+/CXCR4+, but not CD34+/VEGFR2+ PC counts, had similar associations with mortality. Results were validated in a separate cohort of 238 patients with ACS.
CONCLUSIONS: CPC levels are significantly higher in patients after an AMI compared with those with stable CAD and reflect bone marrow PC content. Among patients with ACS, a lower number of hematopoietic-enriched CPCs are associated with a higher mortality.
© 2018 American Heart Association, Inc.

Entities:  

Keywords:  acute coronary syndrome; coronary artery disease; myocardial infarction; non-ST elevated myocardial infarction; prognosis

Mesh:

Substances:

Year:  2018        PMID: 29514830      PMCID: PMC5970041          DOI: 10.1161/CIRCRESAHA.118.312821

Source DB:  PubMed          Journal:  Circ Res        ISSN: 0009-7330            Impact factor:   17.367


  46 in total

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2.  Endogenous G-CSF and CD34+ cell mobilization after acute myocardial infarction.

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3.  Progenitor cell mobilisation in patients with acute and chronic coronary artery disease.

Authors:  A Gaspardone; F Menghini; V Mazzuca; O Skossyreva; G Barbato; P de Fabritiis
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4.  Expression of VEGFR-2 and AC133 by circulating human CD34(+) cells identifies a population of functional endothelial precursors.

Authors:  M Peichev; A J Naiyer; D Pereira; Z Zhu; W J Lane; M Williams; M C Oz; D J Hicklin; L Witte; M A Moore; S Rafii
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6.  Mobilization of endothelial progenitor cells in patients with acute myocardial infarction.

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