| Literature DB >> 29447692 |
Abstract
Related works in this issue of Cell Host & Microbe (Bronner et al., 2018) and in a recent issue of Cell Reports (Hiyoshi et al., 2018) demonstrate how loss-of-function mutations in butyrate utilization and lipopolysaccharide O-antigen processing contribute to evasion of innate host defenses and the convergent evolution of distinct typhoidal Salmonella lineages.Entities:
Mesh:
Substances:
Year: 2018 PMID: 29447692 PMCID: PMC5978414 DOI: 10.1016/j.chom.2018.01.009
Source DB: PubMed Journal: Cell Host Microbe ISSN: 1931-3128 Impact factor: 21.023