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11 in total

Review 1. Behavior, stress, and variability.

Authors: P A Parsons
Journal: Behav Genet Date: 1988-05 Impact factor: 2.805

2. Ethanol-induced formation of colorectal tumours and precursors in a mouse model of Lynch syndrome.

Authors: Guia Cerretelli; Ying Zhou; Mike F Müller; David J Adams; Mark J Arends
Journal: J Pathol Date: 2021-10-13 Impact factor: 9.883

3. Genetic regulation of gene-specific mRNA by ethanol in vivo and its possible role in ethanol preference in a cross with RI lines in mice.

Authors: C E Tagliabracci; S M Singh
Journal: Biochem Genet Date: 1996-06 Impact factor: 1.890

Review 4. Rodent models of alcoholic liver disease: of mice and men.

Authors: Elizabeth Brandon-Warner; Laura W Schrum; C Max Schmidt; Iain H McKillop
Journal: Alcohol Date: 2012-09-07 Impact factor: 2.405

5. Genetics of ocular NAD+-dependent alcohol dehydrogenase and aldehyde dehydrogenase in the mouse: evidence for genetic identity with stomach isozymes and localization of Ahd-4 on chromosome 11 near trembler.

Authors: R S Holmes; R A Popp; J L VandeBerg
Journal: Biochem Genet Date: 1988-04 Impact factor: 1.890

6. Alcoholic hepatitis and metabolic disturbance in female mice: a more tractable model than Nrf2 ^-/- animals.

Authors: Lozan Sheriff; Reenam S Khan; Raquel Saborano; Richard Wilkin; Nguyet-Thin Luu; Ulrich L Gunther; Stefan G Hubscher; Philip N Newsome; Patricia F Lalor
Journal: Dis Model Mech Date: 2020-12-29 Impact factor: 5.758

Review 7. A mini-review of the rodent models for alcoholic liver disease: shortcomings, application, and future prospects.

Authors: Shi-Xuan Liu; Yan-Chao Du; Tao Zeng
Journal: Toxicol Res (Camb) Date: 2021-05-10 Impact factor: 3.524

Biochemical and genetic studies on enzymes of alcohol metabolism: the mouse as a model organism for human studies.

Review 1. Behavior, stress, and variability.

2. Ethanol-induced formation of colorectal tumours and precursors in a mouse model of Lynch syndrome.

3. Genetic regulation of gene-specific mRNA by ethanol in vivo and its possible role in ethanol preference in a cross with RI lines in mice.

Review 4. Rodent models of alcoholic liver disease: of mice and men.

5. Genetics of ocular NAD+-dependent alcohol dehydrogenase and aldehyde dehydrogenase in the mouse: evidence for genetic identity with stomach isozymes and localization of Ahd-4 on chromosome 11 near trembler.

6. Alcoholic hepatitis and metabolic disturbance in female mice: a more tractable model than Nrf2 ^-/- animals.

Review 7. A mini-review of the rodent models for alcoholic liver disease: shortcomings, application, and future prospects.

Review 8. Pathophysiological Aspects of Alcohol Metabolism in the Liver.

Review 9. Alcohol as friend or foe in autoimmune diseases: a role for gut microbiome?

Review 10. Rodent Models of Alcoholic Liver Disease: Role of Binge Ethanol Administration.

Biochemical and genetic studies on enzymes of alcohol metabolism: the mouse as a model organism for human studies.

Review 1. Behavior, stress, and variability.

2. Ethanol-induced formation of colorectal tumours and precursors in a mouse model of Lynch syndrome.

3. Genetic regulation of gene-specific mRNA by ethanol in vivo and its possible role in ethanol preference in a cross with RI lines in mice.

Review 4. Rodent models of alcoholic liver disease: of mice and men.

5. Genetics of ocular NAD+-dependent alcohol dehydrogenase and aldehyde dehydrogenase in the mouse: evidence for genetic identity with stomach isozymes and localization of Ahd-4 on chromosome 11 near trembler.

6. Alcoholic hepatitis and metabolic disturbance in female mice: a more tractable model than Nrf2 -/- animals.

Review 7. A mini-review of the rodent models for alcoholic liver disease: shortcomings, application, and future prospects.

Review 8. Pathophysiological Aspects of Alcohol Metabolism in the Liver.

Review 9. Alcohol as friend or foe in autoimmune diseases: a role for gut microbiome?

Review 10. Rodent Models of Alcoholic Liver Disease: Role of Binge Ethanol Administration.

6. Alcoholic hepatitis and metabolic disturbance in female mice: a more tractable model than Nrf2 ^-/- animals.