Literature DB >> 29338478

Gene-gene-environment interactions of prenatal exposed to environmental tobacco smoke, CYP1A1 and GSTs polymorphisms on full-term low birth weight: relationship of maternal passive smoking, gene polymorphisms, and FT-LBW.

Lihua Huang1, Yijuan Luo1,2, Xiaozhong Wen3,4, Yan-Hui He1, Peng Ding1, Chuanbo Xie1, Tao Liu1, Shi-Xin Yuan5, De-Qin Jia6, Wei-Qing Chen1,7.   

Abstract

OBJECTIVE: To examine the interaction effects of prenatal exposed to environmental tobacco smoke (ETS) and genotypes of cytochrome P4501A1 (CYP1A1), glutathione S-transferases (GSTs) on the risk of full-term low birth weight (FT-LBW). STUDY
DESIGN: We conducted a case-control study among pregnant women at two Women and Children's Hospitals in Guangdong, China (n = 910). Information was collected through interview, medical records review, and blood lab tests. Maternal selfreport and serum cotinine concentration were combined to define prenatal exposed to ETS. Logistic regression approach was applied for statistical analysis.
RESULTS: Our results showed that regardless of genotypes, prenatal exposed to ETS significantly increased the risk of FT-LBW. Then, two-way interactions showed increased prevalence of FT-LBW in prenatal exposed to ETS mothers with the CYP1A1 variant genotype (MspI "CC"), or with GSTT1-null genotype. Furthermore, three-way interactions showed that women with CYP1A1 variant (MspI "TC" or BsrDI "AG") genotypes and GSTT1 "null" genotype had higher risk to give birth of FT-LBW. Additionally, among nonexposed ETS mothers, genotype did not independently confer adverse effects on FT-LBW.
CONCLUSIONS: Our results revealed that prenatal exposed to ETS is independently associated with FT-LBW while gene polymorphisms of CYP1A1 and GSTs merely play modified roles in this process. This study extends understanding of three-way interaction, and stresses the need to tobacco control toward pregnant women for better pregnant outcomes.

Entities:  

Keywords:  Gene polymorphisms; environmental exposure; full-term low birth weight; interaction; smoke; tobacco

Mesh:

Substances:

Year:  2018        PMID: 29338478     DOI: 10.1080/14767058.2018.1429394

Source DB:  PubMed          Journal:  J Matern Fetal Neonatal Med        ISSN: 1476-4954


  2 in total

1.  The discovery BPD (D-BPD) program: study protocol of a prospective translational multicenter collaborative study to investigate determinants of chronic lung disease in very low birth weight infants.

Authors:  Gaston Ofman; Mauricio T Caballero; Damian Alvarez Paggi; Jacqui Marzec; Florencia Nowogrodzki; Hye-Youn Cho; Mariana Sorgetti; Guillermo Colantonio; Alejandra Bianchi; Luis M Prudent; Nestor Vain; Gonzalo Mariani; Jorge Digregorio; Elba Lopez Turconi; Cristina Osio; Fernanda Galletti; Mariangeles Quiros; Andrea Brum; Santiago Lopez Garcia; Silvia Garcia; Douglas Bell; Marcus H Jones; Trent E Tipple; Steven R Kleeberger; Fernando P Polack
Journal:  BMC Pediatr       Date:  2019-07-06       Impact factor: 2.125

2.  Maternal Secondhand Smoke Exposure Enhances Macrosomia Risk Among Pregnant Women Exposed to PM2.5: A New Interaction of Two Air Pollutants in a Nationwide Cohort.

Authors:  Yunyun Luo; Yuelun Zhang; Hui Pan; Shi Chen
Journal:  Front Public Health       Date:  2021-11-18
  2 in total

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