Literature DB >> 29315608

Clinical and research strategies for limb-girdle congenital myasthenic syndromes.

Emily O'Connor1, Ana Töpf1, René P Zahedi2, Sally Spendiff1, Daniel Cox1, Andreas Roos1,2, Hanns Lochmüller1.   

Abstract

Congenital myasthenic syndromes (CMS) are a group of rare disorders that cause fatigable muscle weakness due to defective signal transmission at the neuromuscular junction, a specialized synapse between peripheral motor neurons and their target muscle fibers. There are now over 30 causative genes that have been reported for CMS. Of these, there are 10 that are associated with a limb-girdle pattern of muscle weakness and are thus classed as LG-CMS. Next-generation sequencing and advanced methods of data sharing are likely to uncover further genes that are associated with similar clinical phenotypes, contributing to better diagnosis and effective treatment of LG-CMS patients. This review highlights clinical and pathological hallmarks of LG-CMS in relation to the underlying genetic defects and pathways. Tailored animal and cell models are essential to elucidate the exact function and pathomechanisms at the neuromuscular synapse that underlie LG-CMS. The integration of genomics and proteomics data derived from these models and patients reveals new and often unexpected insights that are relevant beyond the rare genetic disorder of LG-CMS and may extend to the functioning of mammalian synapses in health and disease more generally.
© 2018 New York Academy of Sciences.

Entities:  

Keywords:  animal models; congenital myasthenic syndromes; glycosylation disorders; neuromuscular transmission

Mesh:

Year:  2018        PMID: 29315608     DOI: 10.1111/nyas.13520

Source DB:  PubMed          Journal:  Ann N Y Acad Sci        ISSN: 0077-8923            Impact factor:   5.691


  6 in total

Review 1.  The role of protein glycosylation in muscle diseases.

Authors:  Kai Dang; Shanfeng Jiang; Yuan Gao; Airong Qian
Journal:  Mol Biol Rep       Date:  2022-04-15       Impact factor: 2.742

2.  Congenital myasthenic syndromes in adult neurology clinic: A long road to diagnosis and therapy.

Authors:  Justin C Kao; Margherita Milone; Duygu Selcen; Xin-Ming Shen; Andrew G Engel; Teerin Liewluck
Journal:  Neurology       Date:  2018-10-05       Impact factor: 9.910

3.  Motor Endplate-Anatomical, Functional, and Molecular Concepts in the Historical Perspective.

Authors:  Rüdiger Rudolf; Muzamil Majid Khan; Veit Witzemann
Journal:  Cells       Date:  2019-04-27       Impact factor: 6.600

4.  Modulation of Agrin and RhoA Pathways Ameliorates Movement Defects and Synapse Morphology in MYO9A-Depleted Zebrafish.

Authors:  Emily O'Connor; George Cairns; Sally Spendiff; David Burns; Stefan Hettwer; Armin Mäder; Juliane Müller; Rita Horvath; Clarke Slater; Andreas Roos; Hanns Lochmüller
Journal:  Cells       Date:  2019-08-07       Impact factor: 6.600

5.  Novel NtA and LG1 Mutations in Agrin in a Single Patient Causes Congenital Myasthenic Syndrome.

Authors:  Aiping Wang; Yangyang Xiao; Peng Huang; Lingjuan Liu; Jie Xiong; Jian Li; Ding'an Mao; Liqun Liu
Journal:  Front Neurol       Date:  2020-04-09       Impact factor: 4.003

6.  Clinico-genetic spectrum of limb-girdle muscular weakness in Austria: A multicentre cohort study.

Authors:  Martin Krenn; Matthias Tomschik; Matias Wagner; Gudrun Zulehner; Rosa Weng; Jakob Rath; Sigrid Klotz; Ellen Gelpi; Gabriel Bsteh; Omar Keritam; Isabella Colonna; Chiara Paternostro; Fiona Jäger; Elisabeth Lindeck-Pozza; Stephan Iglseder; Susanne Grinzinger; Martina Schönfelder; Christina Hohenwarter; Manfred Freimüller; Norbert Embacher; Julia Wanschitz; Raffi Topakian; Ana Töpf; Volker Straub; Stefan Quasthoff; Fritz Zimprich; Wolfgang N Löscher; Hakan Cetin
Journal:  Eur J Neurol       Date:  2022-03-10       Impact factor: 6.288

  6 in total

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