| Literature DB >> 29315555 |
Kazuhisa Furue1, Takamichi Ito1, Gaku Tsuji1, Takafumi Kadono2, Takeshi Nakahara1,3, Masutaka Furue1,3.
Abstract
Psoriasis is characterized by widespread scaly erythematous plaques that cause significant physical and psychological burdens for the affected individuals. Accelerated inflammation driven by the tumour necrosis factor-α/interleukin-23/interleukin-17 axis is now known to be the major mechanism in the development of psoriasis. In addition, psoriasis has an autoimmune nature that manifests as autoreactive T cells and is co-morbid with other autoimmune diseases, such as autoimmune bullous diseases, vitiligo, alopecia and thyroiditis. In this article, we review the recent topics on autoimmunity and autoimmune co-morbidities in psoriasis.Entities:
Keywords: alopecia; autoimmunity; bullous pemphigoid; psoriasis; thyroiditis; vitiligo
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Year: 2018 PMID: 29315555 PMCID: PMC5904708 DOI: 10.1111/imm.12891
Source DB: PubMed Journal: Immunology ISSN: 0019-2805 Impact factor: 7.397