Literature DB >> 29287006

Hyperadiponectinemia During Infliximab Induction Therapy in Pediatric Crohn Disease.

Klara Frivolt1,2, Tobias Schwerd1, Stephanie B Schatz1, Folke Freudenberg1,3, Christine Prell1, Katharina J Werkstetter1, Philip Bufler1, Sibylle Koletzko1.   

Abstract

OBJECTIVES: The inflammatory process in Crohn disease (CD) involves the visceral fat, characterized by adipocyte hyperplasia and altered adipose tissue and serum concentrations of tumor necrosis factor (TNF), leptin, adiponectin and resistin. We investigated the effect of anti-TNF therapy with infliximab (IFX) on serum adipokine levels in pediatric CD.
METHODS: Serum concentrations of resistin (ng/mL), leptin (ng/mL), and total adiponectin (μg/mL) were assessed by enzyme-linked immunosorbent assays (ELISA) in 18 pediatric CD patients (mean age 15.0 ± 1.5 years) before first, second, and fourth IFX infusion (weeks 0, 2, and 14) and compared with baseline values from sex- and BMI-matched healthy controls (HC, mean age 13.4 ± 1.6 years).
RESULTS: At baseline, CD patients (mean age 15.0 ± 1.5 years, 10 of 18 boys) compared with HC (13.4 ± 1.6 years, 7 of 15 boys) had higher resistin levels (median 14.7 ng/mL, range 5.1-50.5 vs 7.3 ng/mL, 0.5-14.5); P = 0.0002). At weeks 2 and 14, resistin decreased to 6.9 ng/mL (2.9-16.8) (P < 0.0001) and 9.2 ng/mL (4.1-20.6; P = 0.0011), respectively. Leptin and adiponectin were comparable between patients and HC at baseline. Leptin increased in girls from 9.5 ng/mL (4.0-30.1) to 16.0 ng/mL (7.9-35.2; P = 0.0156) and 17.2 ng/mL (10.8- 26.8; P = 0.1953) at weeks 0, 2, and 14 respectively; with a trend in boys from 2 (0.6-12.9) to 2.8 (1.7-8.6; P = 0.0840) and 3.3 (1.3-4.6; P = 0.1309). Adiponectin peaked initially from 7.8 μg/mL (4.6-11.9) at week 0 to 9.2 μg/mL (4.1-20.7; P = 0.0005) at week 2 and thereafter fell to 6.5 μg/mL (3.0-12.7; P = 0.0182) at week 14.
CONCLUSIONS: TNF blockade is associated with changes in circulating adipokines. The marked early increase of the potent anti-inflammatory adiponectin may contribute to the rapid response to IFX in CD.

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Year:  2018        PMID: 29287006     DOI: 10.1097/MPG.0000000000001876

Source DB:  PubMed          Journal:  J Pediatr Gastroenterol Nutr        ISSN: 0277-2116            Impact factor:   2.839


  5 in total

1.  Glucose and lipid-related biomarkers and the antidepressant response to infliximab in patients with treatment-resistant depression.

Authors:  Mandakh Bekhbat; Karen Chu; Ngoc-Anh Le; Bobbi J Woolwine; Ebrahim Haroon; Andrew H Miller; Jennifer C Felger
Journal:  Psychoneuroendocrinology       Date:  2018-09-06       Impact factor: 4.905

Review 2.  The Role of Adipose Tissue in the Pathogenesis and Therapeutic Outcomes of Inflammatory Bowel Disease.

Authors:  Piotr Eder; Maciej Adler; Agnieszka Dobrowolska; Julian Kamhieh-Milz; Janusz Witowski
Journal:  Cells       Date:  2019-06-21       Impact factor: 6.600

Review 3.  Role of Adipose Tissue in Inflammatory Bowel Disease.

Authors:  Eva Karaskova; Maria Velganova-Veghova; Milos Geryk; Hana Foltenova; Veronika Kucerova; David Karasek
Journal:  Int J Mol Sci       Date:  2021-04-19       Impact factor: 5.923

Review 4.  Dual-therapy strategy for modification of adiponectin receptor signaling in aging-associated chronic diseases.

Authors:  Masaaki Waragai; Gilbert Ho; Yoshiki Takamatsu; Yuka Shimizu; Hiromu Sugino; Shuei Sugama; Takato Takenouchi; Eliezer Masliah; Makoto Hashimoto
Journal:  Drug Discov Today       Date:  2018-05-07       Impact factor: 7.851

5.  Adipokine Resistin Levels at Time of Pediatric Crohn Disease Diagnosis Predict Escalation to Biologic Therapy.

Authors:  Jacob A Kurowski; Jean-Paul Achkar; Rishi Gupta; Iulia Barbur; Tracey L Bonfield; Sarah Worley; Erick M Remer; Claudio Fiocchi; Satish E Viswanath; Marsha H Kay
Journal:  Inflamm Bowel Dis       Date:  2021-06-15       Impact factor: 7.290

  5 in total

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