Literature DB >> 29284828

Aggressive Behavior and Short-Long Polymorphisms of Monoamine Oxidase A: An Example of Effect of Genetic Molecular Mass Change in Psychological Medicine.

Beuy Joob1, Viroj Wiwanitkit2.   

Abstract

Entities:  

Year:  2017        PMID: 29284828      PMCID: PMC5733445          DOI: 10.4103/IJPSYM.IJPSYM_247_17

Source DB:  PubMed          Journal:  Indian J Psychol Med        ISSN: 0253-7176


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Sir, Aggressive behavior is usually an unwanted serious behavior and there are many reports in psychological medicine on the underlying genetic factors. Of several genetic polymorphisms, monoamine oxidase A (MAOA) polymorphism is widely mentioned for the clinical relationship to aggressive behavior.[1] In a recent reported, it was proved that having short polymorphism is related to the increased risk behavior and harmful practice comparing to having long polymorphism.[2] Indeed, based on the basic concern on quantum molecular genetics, the genetic molecular mass change due to different polymorphism can be expected. It is no doubt that the patients with short polymorphism have a less molecular weight of MAOA comparing to those with long polymorphism and this means less catalysis of monoamine can be expected in ones with short polymorphism. Indeed, the high accumulated level of the monoamine is proved to relate to the agitation state in psychiatric patients,[3] and the observation might be well relating to the already mentioned effect of short versus long MAOA polymorphism. The effects of molecular mass change can be well demonstrated for case aggressive behavior and MAOA short and long polymorphism, similar to that seen in other polymorphism related medical disorders.[45]

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Conflicts of interest

There are no conflicts of interest.
  5 in total

1.  Monoamine oxidase and agitation in psychiatric patients.

Authors:  Matea Nikolac Perkovic; Dubravka Svob Strac; Gordana Nedic Erjavec; Suzana Uzun; Josip Podobnik; Oliver Kozumplik; Suzana Vlatkovic; Nela Pivac
Journal:  Prog Neuropsychopharmacol Biol Psychiatry       Date:  2016-02-04       Impact factor: 5.067

2.  Blunted insula activation reflects increased risk and reward seeking as an interaction of testosterone administration and the MAOA polymorphism.

Authors:  Lisa Wagels; Mikhail Votinov; Sina Radke; Benjamin Clemens; Christian Montag; Sonja Jung; Ute Habel
Journal:  Hum Brain Mapp       Date:  2017-06-12       Impact factor: 5.038

3.  Gene-Gene-Environment Interactions of Serotonin Transporter, Monoamine Oxidase A and Childhood Maltreatment Predict Aggressive Behavior in Chinese Adolescents.

Authors:  Yun Zhang; Qing-Sen Ming; Jin-Yao Yi; Xiang Wang; Qiao-Lian Chai; Shu-Qiao Yao
Journal:  Front Behav Neurosci       Date:  2017-02-01       Impact factor: 3.558

4.  HSD11B1 rs846908 polymorphisms and tacrolimus concentrations: quantum chemical analysis and implication in patients with renal transplantation.

Authors:  Beuy Joob; Viroj Wiwanitkit
Journal:  J Nephropharmacol       Date:  2016-01-18

5.  ATP-binding cassette, sub-family B (MDR/TAP), member 1 (ABCB1) polymorphism and clopidogrel concentration in acute coronary syndrome: molecular change can explain the observed therapeutic concentration.

Authors:  Beuy Joob; Viroj Wiwanitkit
Journal:  Anatol J Cardiol       Date:  2016-04       Impact factor: 1.596

  5 in total

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