Literature DB >> 29236891

High expression of XIAP and Bcl-2 may inhibit programmed cell death in glioblastomas.

Daniela Pretti da Cunha Tirapelli1, Isis Lacrose Lustosa1, Sarah Bomfim Menezes1, Indira Maynart Franco1, Andressa Romualdo Rodrigues1, Fernanda Maris Peria2, Alexandre Magno da Nóbrega Marinho3, Luciano Neder Serafini4, Carlos Gilberto Carlotti1, Luís Fernando Tirapelli1.   

Abstract

Glioblastoma (GBM) is the most malignant glioma and represents 29% of all brain tumors. Tumorigenesis is intimately connected with characteristics acquired in the physiologic pathway of cellular death.
OBJECTIVE: In the present study, the expression of anti-apoptotic (XIAP and Bcl-2) and apoptotic (cytochrome C, caspase 9, APAF-1), caspase 3 and the Smac/DIABLO genes related to the apoptosis pathway were evaluated in 30 samples of glioblastoma.
METHODS: The gene expression was evaluated in 30 glioblastomas (WHO grade IV) and compared to 10 white matter control samples with real-time PCR. RESULTS AND
CONCLUSION: There were higher expressions of XIAP (p = 0.0032) and Bcl-2 (p = 0.0351) in the glioblastoma samples compared to the control samples of normal brain. These results raise the question of whether Bcl-2 and XIAP genes can be responsible for the inhibition of programmed cell death in glioblastomas. Moreover, they provide additional information capable of allowing the development of new target therapy strategies.

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Year:  2017        PMID: 29236891     DOI: 10.1590/0004-282X20170156

Source DB:  PubMed          Journal:  Arq Neuropsiquiatr        ISSN: 0004-282X            Impact factor:   1.420


  14 in total

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