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Literature DB >> 29225372

Demystifying Spasticity: Reply to Dietz.

D J Bennett¹.

Abstract

Entities: Disease

Year: 2008 PMID： 29225372 PMCID： PMC5721347 DOI： 10.1152/jn.01279.2007

Source DB: PubMed Journal: J Neurophysiol ISSN： 0022-3077 Impact factor: 2.714

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24 in total

1. Some manifestations of reflex activity in spinal man with particular reference to the occurrence of extensor spasm.

Authors: R A KUHN; M B MACHT
Journal: Bull Johns Hopkins Hosp Date: 1949-01

2. Evaluating the role of botulinum toxin in the management of focal hypertonia in adults.

Authors: D Richardson; G Sheean; D Werring; M Desai; S Edwards; R Greenwood; A Thompson
Journal: J Neurol Neurosurg Psychiatry Date: 2000-10 Impact factor: 10.154

3. Tail muscles become slow but fatigable in chronic sacral spinal rats with spasticity.

Authors: R Luke W Harris; Jacques Bobet; Leo Sanelli; David J Bennett
Journal: J Neurophysiol Date: 2005-11-09 Impact factor: 2.714

4. Motor unit firing during and after voluntary contractions of human thenar muscles weakened by spinal cord injury.

Authors: Inge Zijdewind; Christine K Thomas
Journal: J Neurophysiol Date: 2002-12-11 Impact factor: 2.714

5. Effects of baclofen on spinal reflexes and persistent inward currents in motoneurons of chronic spinal rats with spasticity.

Authors: Y Li; X Li; P J Harvey; D J Bennett
Journal: J Neurophysiol Date: 2004-11 Impact factor: 2.714

Demystifying Spasticity: Reply to Dietz.

1. Some manifestations of reflex activity in spinal man with particular reference to the occurrence of extensor spasm.

2. Evaluating the role of botulinum toxin in the management of focal hypertonia in adults.

3. Tail muscles become slow but fatigable in chronic sacral spinal rats with spasticity.

4. Motor unit firing during and after voluntary contractions of human thenar muscles weakened by spinal cord injury.

5. Effects of baclofen on spinal reflexes and persistent inward currents in motoneurons of chronic spinal rats with spasticity.

6. Spasticity in rats with sacral spinal cord injury.

7. Persistent sodium and calcium currents cause plateau potentials in motoneurons of chronic spinal rats.

8. Role of motoneurons in the generation of muscle spasms after spinal cord injury.

9. Role of endogenous release of norepinephrine in muscle spasms after chronic spinal cord injury.

10. Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.