| Literature DB >> 29212026 |
Kyunghee Noh1, Lingegowda S Mangala2, Hee-Dong Han3, Ningyan Zhang4, Sunila Pradeep5, Sherry Y Wu6, Shaolin Ma6, Edna Mora7, Rajesha Rupaimoole6, Dahai Jiang2, Yunfei Wen6, Mian M K Shahzad6, Yasmin Lyons6, MinSoon Cho8, Wei Hu6, Archana S Nagaraja6, Monika Haemmerle6, Celia S L Mak6, Xiuhui Chen6, Kshipra M Gharpure6, Hui Deng4, Wei Xiong4, Charles V Kingsley9, Jinsong Liu10, Nicholas Jennings6, Michael J Birrer11, Richard R Bouchard9, Gabriel Lopez-Berestein12, Robert L Coleman6, Zhiqiang An4, Anil K Sood13.
Abstract
Angiogenesis inhibitors are important for cancer therapy, but clinically approved anti-angiogenic agents have shown only modest efficacy and can compromise wound healing. This necessitates the development of novel anti-angiogenesis therapies. Here, we show significantly increased EGFL6 expression in tumor versus wound or normal endothelial cells. Using a series of in vitro and in vivo studies with orthotopic and genetically engineered mouse models, we demonstrate the mechanisms by which EGFL6 stimulates tumor angiogenesis. In contrast to its antagonistic effects on tumor angiogenesis, EGFL6 blockage did not affect normal wound healing. These findings have significant implications for development of anti-angiogenesis therapies.Entities:
Keywords: chitosan nanoparticles; ovarian cancer; tumor endothelial cells; tumor vasculature; wound healing
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Year: 2017 PMID: 29212026 PMCID: PMC5749980 DOI: 10.1016/j.celrep.2017.11.020
Source DB: PubMed Journal: Cell Rep Impact factor: 9.423