Literature DB >> 29187090

Outcomes of Patients With Familial Transthyretin Amyloidosis After Liver Transplantation.

Dipti Banerjee1, Lindsey E Roeker2, Martha Grogan3, Paul Swiecicki4, John Poterucha5, Julie Heimbach6, Steve Zeldenrust7, Morie Gertz7, Brooks Edwards3, Richard Daly3, Kyle W Klarich3, Angela Dispenzieri7.   

Abstract

BACKGROUND: Familial transthyretin amyloidosis is a disease caused by misfolded transthyretin aggregates that can impair multiple organ systems. Liver transplantation is the first-line treatment for familial transthyretin amyloidosis. RESEARCH QUESTION: Our objective is to study outcomes and survival among patients with familial transthyretin amyloidosis after transplantation.
DESIGN: All patients undergoing orthotopic liver transplant for familial transthyretin amyloidosis at Mayo Clinic between 1997 and 2012 were reviewed. Baseline clinical characteristics, organs transplanted, and posttransplant clinical course were assessed.
RESULTS: Of the 40 patients, 7 patients had the V30M mutation and 33 had other mutations. Nineteen patients received liver only, 19 liver and heart, and 2 combined liver, heart, and kidney transplants. The 5-year overall survival was 85% for those receiving multiple organ transplant and 52% for those receiving liver transplant only ( P = .057). There was no difference in overall survival based on mutation (V30M vs other mutations), but survival was confounded by varied disease involvement and organs transplanted. Those who had early death (≤24 months from liver transplant) had a higher incidence of baseline peripheral neuropathy, autonomic neuropathy, lower modified BMI, and higher alkaline phosphatase. DISCUSSION: Outcomes of orthotopic liver transplant in familial transthyretin amyloidosis are variable due to heterogeneity in mutations and patient status at the time of transplant. Familial transthyretin amyloidosis can progress, despite liver transplantation. Patients receiving combined liver, heart/kidney transplant demonstrated improved survival compared to liver transplant alone.

Entities:  

Keywords:  heart transplant recipient; hemic and lymphatic diseases; kidney transplant recipient; liver transplant recipient; pathological conditions; signs and symptoms

Mesh:

Year:  2017        PMID: 29187090     DOI: 10.1177/1526924817715463

Source DB:  PubMed          Journal:  Prog Transplant        ISSN: 1526-9248            Impact factor:   1.187


  5 in total

Review 1.  Small Bowel Amyloidosis.

Authors:  Raghav Bansal; Umer Syed; Jacob Walfish; Joshua Aron; Aaron Walfish
Journal:  Curr Gastroenterol Rep       Date:  2018-03-26

Review 2.  Advances in the treatment of hereditary transthyretin amyloidosis: A review.

Authors:  Morie A Gertz; Michelle L Mauermann; Martha Grogan; Teresa Coelho
Journal:  Brain Behav       Date:  2019-08-01       Impact factor: 2.708

Review 3.  Emerging Concepts and Challenges in Rheumatoid Arthritis Gene Therapy.

Authors:  Andrei A Deviatkin; Yulia A Vakulenko; Ludmila V Akhmadishina; Vadim V Tarasov; Marina I Beloukhova; Andrey A Zamyatnin; Alexander N Lukashev
Journal:  Biomedicines       Date:  2020-01-09

Review 4.  Functional Interactions Between lncRNAs/circRNAs and miRNAs: Insights Into Rheumatoid Arthritis.

Authors:  Juan-Juan Han; Xue-Qiang Wang; Xin-An Zhang
Journal:  Front Immunol       Date:  2022-02-07       Impact factor: 7.561

Review 5.  Amyloid seeding as a disease mechanism and treatment target in transthyretin cardiac amyloidosis.

Authors:  Paolo Morfino; Alberto Aimo; Giorgia Panichella; Claudio Rapezzi; Michele Emdin
Journal:  Heart Fail Rev       Date:  2022-04-06       Impact factor: 4.654

  5 in total

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