Literature DB >> 29180494

Metabolism: A Direct Link Between Cardiac Structure and Function.

Paul S Brookes1, Heinrich Taegtmeyer2.   

Abstract

Entities:  

Keywords:  Editorials; hypertrophy; metabolism; metabolomics

Mesh:

Substances:

Year:  2017        PMID: 29180494      PMCID: PMC5785082          DOI: 10.1161/CIRCULATIONAHA.117.031372

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


× No keyword cloud information.
  14 in total

Review 1.  Myocardial substrate metabolism in the normal and failing heart.

Authors:  William C Stanley; Fabio A Recchia; Gary D Lopaschuk
Journal:  Physiol Rev       Date:  2005-07       Impact factor: 37.312

Review 2.  Cardiac plasticity.

Authors:  Joseph A Hill; Eric N Olson
Journal:  N Engl J Med       Date:  2008-03-27       Impact factor: 91.245

3.  Cardiac-specific overexpression of GLUT1 prevents the development of heart failure attributable to pressure overload in mice.

Authors:  Ronglih Liao; Mohit Jain; Lei Cui; Jessica D'Agostino; Francesco Aiello; Ivan Luptak; Soeun Ngoy; Richard M Mortensen; Rong Tian
Journal:  Circulation       Date:  2002-10-15       Impact factor: 29.690

Review 4.  Mitochondrial adaptations to physiological vs. pathological cardiac hypertrophy.

Authors:  E Dale Abel; Torsten Doenst
Journal:  Cardiovasc Res       Date:  2011-01-21       Impact factor: 10.787

Review 5.  O-GlcNAc and the cardiovascular system.

Authors:  Sujith Dassanayaka; Steven P Jones
Journal:  Pharmacol Ther       Date:  2013-11-25       Impact factor: 12.310

6.  Cardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxia.

Authors:  Qianwen Wang; Rajakumar V Donthi; Jianxun Wang; Alex J Lange; Lewis J Watson; Steven P Jones; Paul N Epstein
Journal:  Am J Physiol Heart Circ Physiol       Date:  2008-05-02       Impact factor: 4.733

7.  Cardiac expression of kinase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase inhibits glycolysis, promotes hypertrophy, impairs myocyte function, and reduces insulin sensitivity.

Authors:  Rajakumar V Donthi; Gang Ye; Chaodong Wu; Donald A McClain; Alex J Lange; Paul N Epstein
Journal:  J Biol Chem       Date:  2004-08-25       Impact factor: 5.157

Review 8.  Oncometabolites: tailoring our genes.

Authors:  Stefan Nowicki; Eyal Gottlieb
Journal:  FEBS J       Date:  2015-04-30       Impact factor: 5.542

9.  Glucose regulation of load-induced mTOR signaling and ER stress in mammalian heart.

Authors:  Shiraj Sen; Bijoy K Kundu; Henry Cheng-Ju Wu; S Shahrukh Hashmi; Patrick Guthrie; Landon W Locke; R Jack Roy; G Paul Matherne; Stuart S Berr; Matthew Terwelp; Brian Scott; Sylvia Carranza; O Howard Frazier; David K Glover; Wolfgang H Dillmann; Michael J Gambello; Mark L Entman; Heinrich Taegtmeyer
Journal:  J Am Heart Assoc       Date:  2013-05-17       Impact factor: 5.501

10.  Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac Growth.

Authors:  Andrew A Gibb; Paul N Epstein; Shizuka Uchida; Yuting Zheng; Lindsey A McNally; Detlef Obal; Kartik Katragadda; Patrick Trainor; Daniel J Conklin; Kenneth R Brittian; Michael T Tseng; Jianxun Wang; Steven P Jones; Aruni Bhatnagar; Bradford G Hill
Journal:  Circulation       Date:  2017-08-31       Impact factor: 29.690

View more
  3 in total

Review 1.  Metabolic Coordination of Physiological and Pathological Cardiac Remodeling.

Authors:  Andrew A Gibb; Bradford G Hill
Journal:  Circ Res       Date:  2018-06-22       Impact factor: 17.367

2.  PHDs/CPT1B/VDAC1 axis regulates long-chain fatty acid oxidation in cardiomyocytes.

Authors:  Aude Angelini; Pradip K Saha; Antrix Jain; Sung Yun Jung; Randall L Mynatt; Xinchun Pi; Liang Xie
Journal:  Cell Rep       Date:  2021-10-05       Impact factor: 9.423

Review 3.  Metabolic Mechanisms of Exercise-Induced Cardiac Remodeling.

Authors:  Kyle Fulghum; Bradford G Hill
Journal:  Front Cardiovasc Med       Date:  2018-09-11
  3 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.