Literature DB >> 29163965

Use of inhibitors of the renin-angiotensin system is associated with longer survival in patients with hepatocellular carcinoma.

Matthias Pinter1,2, Arndt Weinmann3,4,5, Marcus-Alexander Wörns3,4, Florian Hucke1,6, Simona Bota1,6, Jens U Marquardt3,4, Dan G Duda2, Rakesh K Jain2, Peter R Galle3,4, Michael Trauner1, Markus Peck-Radosavljevic1,6, Wolfgang Sieghart1.   

Abstract

BACKGROUND: Inhibition of the renin-angiotensin system (RAS) was associated with longer survival in patients with different solid malignancies.
OBJECTIVE: The objective of this study was to investigate the effect of RAS inhibitor (RASi) treatment (angiotensin-converting enzyme inhibitors or angiotensin-II-receptor blockers) on survival of patients with hepatocellular carcinoma (HCC).
METHODS: Patients diagnosed with HCC and Child-Pugh A between 1992 and 2013 who received sorafenib, experimental therapy, or best supportive care were eligible for the Vienna cohort. The Mainz cohort included patients with HCC and Child-Pugh A who received sorafenib treatment between 2007 and 2016. The association between RASi and overall survival (OS) was evaluated in univariate and multivariate analyses.
RESULTS: In the Vienna cohort, 43 of 156 patients received RASi for hypertension. RASi treatment was associated with longer OS (11.9 vs. 6.8 months (mo); p = 0.014) and remained a significant prognostic factor upon multivariate analysis (HR = 0.6; 95% CI 0.4-0.9; p = 0.011). In subgroup analysis, patients treated with sorafenib plus RASi had better median OS (19.5 mo) compared to those treated with either sorafenib (10.9 mo) or RASi (9.7 mo) alone (p = 0.043). The beneficial effect of RASi on survival was confirmed in the Mainz cohort (n = 76).
CONCLUSION: RAS inhibition is associated with longer survival in HCC patients with Child-Pugh class A.

Entities:  

Keywords:  Hepatocellular carcinoma; angiotensin II receptor blocker; angiotensin-converting enzyme inhibitor; renin–angiotensin system; sorafenib

Year:  2017        PMID: 29163965      PMCID: PMC5676550          DOI: 10.1177/2050640617695698

Source DB:  PubMed          Journal:  United European Gastroenterol J        ISSN: 2050-6406            Impact factor:   4.623


  68 in total

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