Literature DB >> 29110849

Continued use of afatinib with the addition of cetuximab after progression on afatinib in patients with EGFR mutation-positive non-small-cell lung cancer and acquired resistance to gefitinib or erlotinib.

Leora Horn1, Scott Gettinger2, D Ross Camidge3, Egbert F Smit4, Yelena Y Janjigian5, Vincent A Miller6, William Pao7, Matthias Freiwald8, Jean Fan9, Bushi Wang10, Vikram K Chand11, Harry J M Groen12.   

Abstract

OBJECTIVES: In a phase Ib trial, afatinib plus cetuximab demonstrated promising clinical activity (objective response rate [ORR]: 29%; median progression-free survival [PFS]: 4.7 months) in patients with epidermal growth factor receptor (EGFR) mutation-positive non-small-cell lung cancer (NSCLC) with acquired resistance to erlotinib or gefitinib. Here, a separate cohort exploring afatinib plus cetuximab after progression on afatinib is reported.
MATERIALS AND METHODS: Patients with EGFR mutation-positive NSCLC who progressed on erlotinib or gefitinib received afatinib 40mg daily until progression, followed by afatinib daily plus cetuximab 500mg/m2 every 2 weeks until progression or intolerable adverse events (AEs). Endpoints included safety, ORR, and PFS.
RESULTS: Thirty-seven patients received afatinib monotherapy. Two (5%) patients responded; median PFS was 2.7 months. Thirty-six patients transitioned to afatinib plus cetuximab. Four (11%) patients responded; median PFS was 2.9 months. Median PFS with afatinib plus cetuximab for patients who received afatinib monotherapy for ≥12 versus <12 weeks was 4.9 versus 1.8 months (p=0.0354), and for patients with T790M-positive versus T790M-negative tumors was 4.8 versus 1.8 months (p=0.1306). Fifty percent of patients receiving afatinib plus cetuximab experienced drug-related grade 3/4 AEs. The most frequent drug-related AEs (any grade) were diarrhea (70%), rash (49%), and fatigue (35%) with afatinib monotherapy and rash (69%), paronychia (39%), and dry skin (36%) with afatinib plus cetuximab.
CONCLUSION: Sequential EGFR blockade with afatinib followed by afatinib plus cetuximab had a predictable safety profile and demonstrated modest activity in patients with EGFR mutation-positive NSCLC with resistance to erlotinib or gefitinib. CLINICALTRIALS. GOV IDENTIFIER: NCT01090011.
Copyright © 2017 The Author(s). Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Afatinib; Cetuximab; EGFR; NSCLC; Phase Ib

Mesh:

Substances:

Year:  2017        PMID: 29110849     DOI: 10.1016/j.lungcan.2017.08.014

Source DB:  PubMed          Journal:  Lung Cancer        ISSN: 0169-5002            Impact factor:   5.705


  6 in total

1.  Diverse EGFR Exon 20 Insertions and Co-Occurring Molecular Alterations Identified by Comprehensive Genomic Profiling of NSCLC.

Authors:  Jonathan W Riess; David R Gandara; Garrett M Frampton; Russell Madison; Nir Peled; Jose A Bufill; Grace K Dy; Sai-Hong Ignatius Ou; Philip J Stephens; John D McPherson; Primo N Lara; Rebekah A Burich; Jeffrey S Ross; Vincent A Miller; Siraj M Ali; Philip C Mack; Alexa B Schrock
Journal:  J Thorac Oncol       Date:  2018-07-05       Impact factor: 15.609

2.  Structure-function analysis of oncogenic EGFR Kinase Domain Duplication reveals insights into activation and a potential approach for therapeutic targeting.

Authors:  Zhenfang Du; Benjamin P Brown; Soyeon Kim; Donna Ferguson; Dean C Pavlick; Gowtham Jayakumaran; Ryma Benayed; Jean-Nicolas Gallant; Yun-Kai Zhang; Yingjun Yan; Monica Red-Brewer; Siraj M Ali; Alexa B Schrock; Ahmet Zehir; Marc Ladanyi; Adam W Smith; Jens Meiler; Christine M Lovly
Journal:  Nat Commun       Date:  2021-03-02       Impact factor: 14.919

Review 3.  The Right Partner in Crime: Unlocking the Potential of the Anti-EGFR Antibody Cetuximab via Combination With Natural Killer Cell Chartering Immunotherapeutic Strategies.

Authors:  Hasan Baysal; Ines De Pauw; Hannah Zaryouh; Marc Peeters; Jan Baptist Vermorken; Filip Lardon; Jorrit De Waele; An Wouters
Journal:  Front Immunol       Date:  2021-09-07       Impact factor: 7.561

Review 4.  Advances in targeting acquired resistance mechanisms to epidermal growth factor receptor tyrosine kinase inhibitors.

Authors:  Justin A Chen; Jonathan W Riess
Journal:  J Thorac Dis       Date:  2020-05       Impact factor: 3.005

5.  Dual EGFR blockade with cetuximab and erlotinib combined with anti-VEGF antibody bevacizumab in advanced solid tumors: a phase 1 dose escalation triplet combination trial.

Authors:  Vivek Subbiah; Ecaterina Ileana Dumbrava; Razelle Kurzrock; Gerald Falchook; Yunfang Jiang; Kyaw Z Thein; Aung Naing; David S Hong; Siqing Fu; Sarina A Piha-Paul; Apostolia M Tsimberidou; Filip Janku; Funda Meric-Bernstam
Journal:  Exp Hematol Oncol       Date:  2020-04-20

6.  EGFR-Specific Tyrosine Kinase Inhibitor Modifies NK Cell-Mediated Antitumoral Activity against Ovarian Cancer Cells.

Authors:  Nina Mallmann-Gottschalk; Yvonne Sax; Rainer Kimmig; Stephan Lang; Sven Brandau
Journal:  Int J Mol Sci       Date:  2019-09-22       Impact factor: 5.923

  6 in total

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