Literature DB >> 29103763

Occurrence of metachronous or synchronous lesions after endoscopic treatment of gastric epithelia dysplasia- impact of histologic features of background mucosa.

Won-Young Park1, So-Jeong Lee1, Young-Keum Kim1, Ahrong Kim1, Do Youn Park2, Bong-Eun Lee3, Geun-Am Song3, Gwang Ha Kim3.   

Abstract

AIMS: Endoscopic resection is a safe and effective method to treat gastric epithelia dysplasia (GED). However, the development of metachronous and synchronous lesions after treatment has become a major concern. In this study, we investigated clinicopathologic features of 105 GED lesions from endoscopic resections between January 2008 and December 2009. Our goal is to find histologic factors that predict synchronous and metachronous lesions after ESD treatment. We assessed the degree of intestinal metaplasia (IM) and atrophy, type of IM, presence of gastritis cystica profunda, and crypt dysplasia in the adjacent mucosa. METHODS AND
RESULTS: We divided 105 GED lesions into three groups: a single group without metachronous or synchronous GED or adenocarcinoma (n=35); a multiple synchronous group (n=30, group with synchronous occurrence of GED or adenocarcinoma after treatment); and a multiple metachronous group (n=40, group with metachronous occurrence of GED or adenocarcinoma after treatment). The multiple metachronous and synchronous groups showed larger sizes (p=0.003) and higher grades (p=0.021) as compared with the single group. Furthermore, marked IM and atrophy in adjacent mucosa were more easily seen in the multiple metachronous and synchronous groups as compared with the single group (p<0.0001). Interestingly, the presence of incomplete type of IM (p=0.025) and crypt dysplasia (p<0.0001) in background mucosa was associated with occurrence of metachronous and synchronous lesions following endoscopic resection of GED.
CONCLUSIONS: The histological features of background mucosa, such as intestinal metaplasia, atrophy, and crypt dysplasia could be used as indicators of occurrence of metachronous and synchronous lesions after endoscopic treatment of GED.
Copyright © 2017 Elsevier GmbH. All rights reserved.

Entities:  

Keywords:  Atrophy; Crypt dysplasia; Gastric epithelial dysplasia; Intestinal metaplasia; Multiple lesions; Stomach

Mesh:

Year:  2017        PMID: 29103763     DOI: 10.1016/j.prp.2017.10.022

Source DB:  PubMed          Journal:  Pathol Res Pract        ISSN: 0344-0338            Impact factor:   3.250


  4 in total

Review 1.  Brunner's Gland Hamartoma of the Duodenum: A Literature Review.

Authors:  Menghua Zhu; Hongyu Li; Yanyan Wu; Yang An; Yuye Wang; Chun Ye; Dan Zhang; Rui Ma; Xuehan Wang; Xiaodong Shao; Xiaozhong Guo; Xingshun Qi
Journal:  Adv Ther       Date:  2021-04-29       Impact factor: 3.845

2.  Computed tomography features and clinical characteristics of gastritis cystica profunda.

Authors:  Rui Wang; Hao Lu; Juan Yu; Wenpeng Huang; Jing Li; Ming Cheng; Pan Liang; Liming Li; Huiping Zhao; Jianbo Gao
Journal:  Insights Imaging       Date:  2022-01-24

3.  Clinical outcomes of metachronous recurrence of gastric epithelial neoplasia based on Helicobacter pylori infection status and microsatellite stability.

Authors:  Yeon-Ji Kim; Jaeyoung Kim; Woo Chul Chung
Journal:  Korean J Intern Med       Date:  2022-05-04       Impact factor: 3.165

4.  Long-term outcomes of patients with gastric adenoma in Korea: A retrospective observational study.

Authors:  Tae Young Park; Su Jin Jeong; Tae Hyung Kim; Jin Lee; Jongha Park; Tae Oh Kim; Yong Eun Park
Journal:  Medicine (Baltimore)       Date:  2020-03       Impact factor: 1.817

  4 in total

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