Literature DB >> 29069327

The role of complement inhibition in kidney transplantation.

C Legendre1,2,3, R Sberro-Soussan1, J Zuber1,2,4, V Frémeaux-Bacchi5,6.   

Abstract

INTRODUCTION AND
BACKGROUND: The complement system which belongs to the innate immune system acts both as a first line of defence against various pathogens and as a guardian of host homeostasis. The role of complement has been recently highlighted in several aspects of kidney transplantation: ischaemia-reperfusion, antibody-mediated rejection and native kidney disease recurrence. SOURCES OF DATA: Experimental data, availability of complement-blocking molecules (mainly the anti-C5 monoclonal antibody, eculizumab) and several trials in human kidney transplant recipients has led to some areas of agreement and some disappointment. AREAS OF AGREEMENT AND CONTROVERSIES: So far, eculizumab has shown great efficacy in treatment and prevention of atypical haemolytic and uraemic syndrome, some efficacy in the prevention of antibody-mediated and so far no efficacy in the prevention of delayed graft function. GROWING POINTS: Among the numerous potentially available drugs potentially interfering with complement, recent focus has been made on C1 blockers in the setting of antibody-mediated rejection with promising results. AREAS TIMELY FOR DEVELOPING RESEARCH: Complement is now recognized as a major player in transplant immunology, several targets are going to be tested to define precisely which ones may be potentially useful in clinical practice.
© The Author 2017. Published by Oxford University Press. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com

Entities:  

Keywords:  antibody-mediated rejection; atypical hemolytic uremic syndrome; complement; eculizumab; ischemia-reperfusion

Mesh:

Substances:

Year:  2017        PMID: 29069327     DOI: 10.1093/bmb/ldx037

Source DB:  PubMed          Journal:  Br Med Bull        ISSN: 0007-1420            Impact factor:   4.291


  5 in total

1.  Antibodies against complement component C5 prevent antibody-mediated rejection after lung transplantation in murine orthotopic models with skin-graft-induced pre-sensitization.

Authors:  Yuki Shiina; Hidemi Suzuki; Atsushi Hata; Taisuke Kaiho; Hiroki Matsumoto; Takahide Toyoda; Yuichi Sakairi; Hironobu Wada; Shinichiro Motohashi; Ichiro Yoshino
Journal:  Gen Thorac Cardiovasc Surg       Date:  2022-06-29

Review 2.  Perspectives on the Optimal Genetically Engineered Pig in 2018 for Initial Clinical Trials of Kidney or Heart Xenotransplantation.

Authors:  David K C Cooper; Mohamed Ezzelarab; Hayato Iwase; Hidetaka Hara
Journal:  Transplantation       Date:  2018-12       Impact factor: 4.939

3.  Complement component C5a induces aberrant epigenetic modifications in renal tubular epithelial cells accelerating senescence by Wnt4/βcatenin signaling after ischemia/reperfusion injury.

Authors:  Giuseppe Castellano; Rossana Franzin; Fabio Sallustio; Alessandra Stasi; Barbara Banelli; Massimo Romani; Giuseppe De Palma; Giuseppe Lucarelli; Chiara Divella; Michele Battaglia; Antonio Crovace; Francesco Staffieri; Giuseppe Grandaliano; Giovanni Stallone; Pasquale Ditonno; Paolo Cravedi; Vincenzo Cantaluppi; Loreto Gesualdo
Journal:  Aging (Albany NY)       Date:  2019-07-08       Impact factor: 5.682

Review 4.  Nanobodies: new avenue to treat kidney disease.

Authors:  Nicola Wanner; Thomas Eden; Nastassia Liaukouskaya; Friedrich Koch-Nolte
Journal:  Cell Tissue Res       Date:  2021-06-16       Impact factor: 4.051

Review 5.  Renal Delivery of Pharmacologic Agents During Machine Perfusion to Prevent Ischaemia-Reperfusion Injury: From Murine Model to Clinical Trials.

Authors:  Rossana Franzin; Alessandra Stasi; Marco Fiorentino; Simona Simone; Rainer Oberbauer; Giuseppe Castellano; Loreto Gesualdo
Journal:  Front Immunol       Date:  2021-07-06       Impact factor: 7.561

  5 in total

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