Literature DB >> 28973341

Edaravone Prevents Retinal Degeneration in Adult Mice Following Optic Nerve Injury.

Goichi Akiyama1, Yuriko Azuchi1, Xiaoli Guo1, Takahiko Noro1, Atsuko Kimura1, Chikako Harada1, Kazuhiko Namekata1, Takayuki Harada1.   

Abstract

Purpose: To assess the therapeutic potential of edaravone, a free radical scavenger that is used for the treatment of acute brain infarction and amyotrophic lateral sclerosis, in a mouse model of optic nerve injury (ONI).
Methods: Two microliters of edaravone (7.2 mM) or vehicle were injected intraocularly 3 minutes after ONI. Optical coherence tomography, retrograde labeling of retinal ganglion cells (RGCs), histopathology, and immunohistochemical analyses of phosphorylated apoptosis signal-regulating kinase-1 (ASK1) and p38 mitogen-activated protein kinase (MAPK) in the retina were performed after ONI. Reactive oxygen species (ROS) levels were assessed with a CellROX Green Reagent.
Results: Edaravone ameliorated ONI-induced ROS production, RGC death, and inner retinal degeneration. Also, activation of the ASK1-p38 MAPK pathway that induces RGC death following ONI was suppressed with edaravone treatment. Conclusions: The results of this study suggest that intraocular administration of edaravone may be a useful treatment for posttraumatic complications.

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Year:  2017        PMID: 28973341     DOI: 10.1167/iovs.17-22250

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  7 in total

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Review 4.  Suppression of Oxidative Stress as Potential Therapeutic Approach for Normal Tension Glaucoma.

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Review 5.  Recent advances in genetically modified animal models of glaucoma and their roles in drug repositioning.

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  7 in total

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