Literature DB >> 28917824

Parkinson's disease susceptibility variants and severity of Lewy body pathology.

Michael G Heckman1, Koji Kasanuki2, Nancy N Diehl3, Shunsuke Koga4, Alexandra Soto5, Melissa E Murray6, Dennis W Dickson7, Owen A Ross8.   

Abstract

INTRODUCTION: Meta-analyses of genome-wide association studies (GWAS) have established common genetic risk factors for clinical Parkinson's disease (PD); however, associations between these risk factors and quantitative neuropathologic markers of disease severity have not been well-studied. This study evaluated associations of nominated variants from the most recent PD GWAS meta-analysis with Lewy body disease (LBD) subtype (brainstem, transitional, or diffuse) and pathologic burden of LB pathology as measured by LB counts in five cortical regions in a series of LBD cases.
METHODS: 547 autopsy-confirmed cases of LBD were included and genotyped for 29 different GWAS-nominated PD risk variants. LB counts were measured in middle frontal (MF), superior temporal (ST), inferior parietal (IP), cingulate (CG), and parahippocampal (PH) gyri.
RESULTS: None of the variants examined were significantly associated with LB counts in any brain region or with LBD subtype after correcting for multiple testing. Nominally significant (P < 0.05) associations with LB counts where the direction of association was in agreement with that observed in the PD GWAS meta-analysis were observed for variants in BCKDK/STX1B (MF, ST, IP) and SNCA (ST). Additionally, MIR4697 and BCKDK/STX1B variants were nominally associated with LBD subtype.
CONCLUSION: The lack of a significant association between PD GWAS variants and severity of LB pathology is consistent with the generally subtle association odds ratios that have been observed in disease-risk analysis. These results also suggest that genetic factors other than the susceptibility loci may determine quantitative neuropathologic outcomes in patients with LBD.
Copyright © 2017 Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Genetics; Lewy body disease; Neuropathology; Parkinson's disease

Mesh:

Year:  2017        PMID: 28917824      PMCID: PMC5716921          DOI: 10.1016/j.parkreldis.2017.09.009

Source DB:  PubMed          Journal:  Parkinsonism Relat Disord        ISSN: 1353-8020            Impact factor:   4.891


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