Literature DB >> 28916543

Cryopyrin-associated Periodic Syndromes in Italian Patients: Evaluation of the Rate of Somatic NLRP3 Mosaicism and Phenotypic Characterization.

Denise Lasigliè1,2, Anna Mensa-Vilaro1,2, Denise Ferrera1,2, Roberta Caorsi1,2, Federica Penco1,2, Giuseppe Santamaria1,2, Marco Di Duca1,2, Giulia Amico1,2, Kenji Nakagawa1,2, Francesca Antonini1,2, Alberto Tommasini1,2, Rita Consolini1,2, Antonella Insalaco1,2, Marco Cattalini1,2, Laura Obici1,2, Romina Gallizzi1,2, Francesca Santarelli1,2, Genny Del Zotto1,2, Mariasavina Severino1,2, Anna Rubartelli1,2, Roberto Ravazzolo1,2, Alberto Martini1,2, Isabella Ceccherini1,2, Ryuta Nishikomori1,2, Marco Gattorno1,2, Juan I Arostegui1,2, Silvia Borghini3,4.   

Abstract

OBJECTIVE: To evaluate the rate of somatic NLRP3 mosaicism in an Italian cohort of mutation-negative patients with cryopyrin-associated periodic syndrome (CAPS).
METHODS: The study enrolled 14 patients with a clinical phenotype consistent with CAPS in whom Sanger sequencing of the NLRP3 gene yielded negative results. Patients' DNA were subjected to amplicon-based NLRP3 deep sequencing.
RESULTS: Low-level somatic NLRP3 mosaicism has been detected in 4 patients, 3 affected with chronic infantile neurological cutaneous and articular syndrome and 1 with Muckle-Wells syndrome. Identified nucleotide substitutions encode for 4 different amino acid exchanges, with 2 of them being novel (p.Y563C and p.G564S). In vitro functional studies confirmed the deleterious behavior of the 4 somatic NLRP3 mutations. Among the different neurological manifestations detected, 1 patient displayed mild loss of white matter volume on brain magnetic resonance imaging.
CONCLUSION: The allele frequency of somatic NLRP3 mutations occurs generally under 15%, considered the threshold of detectability using the Sanger method of DNA sequencing. Consequently, routine genetic diagnostic of CAPS should be currently performed by next-generation techniques ensuring high coverage to identify also low-level mosaicism, whose actual frequency is yet unknown and probably underestimated.

Entities:  

Keywords:  GENETIC STUDIES; INFLAMMATION; NEUROLOGIC MANIFESTATIONS; PEDIATRIC RHEUMATIC DISEASES

Mesh:

Substances:

Year:  2017        PMID: 28916543     DOI: 10.3899/jrheum.170041

Source DB:  PubMed          Journal:  J Rheumatol        ISSN: 0315-162X            Impact factor:   4.666


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