Literature DB >> 28914713

The hyperfibrinolytic phenotype is the most lethal and resource intense presentation of fibrinolysis in massive transfusion patients.

John R Taylor1, Erin E Fox, John B Holcomb, Sandro Rizoli, Kenji Inaba, Martin A Schreiber, Karen Brasel, Thomas M Scalea, Charles E Wade, Eileen Bulger, Bryan A Cotton.   

Abstract

BACKGROUND: Among bleeding patients, we hypothesized that the hyperfibrinolytic (HF) phenotype would be associated with the highest mortality, whereas shutdown (SD) patients would have the greatest complication burden.
METHODS: Severely injured patients predicted to receive a massive transfusion at 12 Level I trauma centers were randomized to one of two transfusion ratios as described in the Pragmatic, Randomized, Optimal Platelet and Plasma Ratio trial. Fibrinolysis phenotypes were determined based on admission clot lysis at 30 minutes (LY30): SD ≤0.8%, physiologic (PHYS) 0.9-2.9%, and HF ≥3%. Univariate and multivariate analysis was performed. Logistic regression was used to adjust for age, gender, arrival physiology, shock, injury severity, center effect, and treatment arm.
RESULTS: Among the 680 patients randomized, 547(80%) had admission thrombelastography (TEG) values available to determine fibrinolytic phenotypes. Compared to SD and PHYS, HF patients had higher Injury Severity Score (25 vs. 25 vs. 34), greater base deficit (-8 vs. -6 vs. -12) and were more uniformly hypocoagulable on admission by PT, PTT, and TEG values; all p <0.001. HF patients also received more red blood cells, plasma, and platelets (at 3, 6, and 24 hours); had fewer ICU-, ventilator-, and hospital-free days; and had higher 24-hour and 30-day mortality. There were no differences in complications between the three phenotypes. Multivariate logistic regression demonstrated that HF on admission was associated with a threefold higher mortality (OR 3.06, 95% CI 1.57-5.95, p = 0.001).
CONCLUSIONS: Previous data have shown that both the SD and HF phenotypes are associated with increased mortality and complications in the general trauma population. However, in a large cohort of bleeding patients, HF was confirmed to be a much more lethal and resource-intense phenotype. These data suggest that further research into the understanding of SD and HF is warranted to improve outcomes in this patient population. LEVEL OF EVIDENCE: Prognostic, level II.

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Year:  2018        PMID: 28914713      PMCID: PMC5739990          DOI: 10.1097/TA.0000000000001699

Source DB:  PubMed          Journal:  J Trauma Acute Care Surg        ISSN: 2163-0755            Impact factor:   3.313


  15 in total

1.  Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.

Authors:  Michael P Chapman; Ernest E Moore; Christopher R Ramos; Arsen Ghasabyan; Jeffrey N Harr; Theresa L Chin; John R Stringham; Angela Sauaia; Christopher C Silliman; Anirban Banerjee
Journal:  J Trauma Acute Care Surg       Date:  2013-12       Impact factor: 3.313

2.  Hyperfibrinolysis at admission is an uncommon but highly lethal event associated with shock and prehospital fluid administration.

Authors:  Bryan A Cotton; John A Harvin; Vadim Kostousouv; Kristin M Minei; Zayde A Radwan; Herbert Schöchl; Charles E Wade; John B Holcomb; Nena Matijevic
Journal:  J Trauma Acute Care Surg       Date:  2012-08       Impact factor: 3.313

3.  Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapy.

Authors:  Hunter B Moore; Ernest E Moore; Eduardo Gonzalez; Michael P Chapman; Theresa L Chin; Christopher C Silliman; Anirban Banerjee; Angela Sauaia
Journal:  J Trauma Acute Care Surg       Date:  2014-12       Impact factor: 3.313

4.  The ratio of blood products transfused affects mortality in patients receiving massive transfusions at a combat support hospital.

Authors:  Matthew A Borgman; Philip C Spinella; Jeremy G Perkins; Kurt W Grathwohl; Thomas Repine; Alec C Beekley; James Sebesta; Donald Jenkins; Charles E Wade; John B Holcomb
Journal:  J Trauma       Date:  2007-10

5.  Early prediction of massive transfusion in trauma: simple as ABC (assessment of blood consumption)?

Authors:  Timothy C Nunez; Igor V Voskresensky; Lesly A Dossett; Ricky Shinall; William D Dutton; Bryan A Cotton
Journal:  J Trauma       Date:  2009-02

6.  The prospective, observational, multicenter, major trauma transfusion (PROMMTT) study: comparative effectiveness of a time-varying treatment with competing risks.

Authors:  John B Holcomb; Deborah J del Junco; Erin E Fox; Charles E Wade; Mitchell J Cohen; Martin A Schreiber; Louis H Alarcon; Yu Bai; Karen J Brasel; Eileen M Bulger; Bryan A Cotton; Nena Matijevic; Peter Muskat; John G Myers; Herb A Phelan; Christopher E White; Jiajie Zhang; Mohammad H Rahbar
Journal:  JAMA Surg       Date:  2013-02       Impact factor: 14.766

7.  Activated thrombin-activatable fibrinolysis inhibitor (TAFIa) levels are decreased in patients with trauma-induced coagulopathy.

Authors:  T Lustenberger; B Relja; B Puttkammer; E C Gabazza; E Geiger; Y Takei; J Morser; I Marzi
Journal:  Thromb Res       Date:  2012-11-20       Impact factor: 3.944

8.  Postinjury multiple organ failure: a bimodal phenomenon.

Authors:  F A Moore; A Sauaia; E E Moore; J B Haenel; J M Burch; D C Lezotte
Journal:  J Trauma       Date:  1996-04

9.  Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients.

Authors:  Michael P Chapman; Ernest E Moore; Hunter B Moore; Eduardo Gonzalez; Fabia Gamboni; James G Chandler; Sanchayita Mitra; Arsen Ghasabyan; Theresa L Chin; Angela Sauaia; Anirban Banerjee; Christopher C Silliman
Journal:  J Trauma Acute Care Surg       Date:  2016-01       Impact factor: 3.313

10.  Traumatic Endotheliopathy: A Prospective Observational Study of 424 Severely Injured Patients.

Authors:  Pär I Johansson; Hanne H Henriksen; Jakob Stensballe; Mikkel Gybel-Brask; Jessica C Cardenas; Lisa A Baer; Bryan A Cotton; John B Holcomb; Charles E Wade; Sisse R Ostrowski
Journal:  Ann Surg       Date:  2017-03       Impact factor: 12.969

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  5 in total

1.  TEG Lysis Shutdown Represents Coagulopathy in Bleeding Trauma Patients: Analysis of the PROPPR Cohort.

Authors:  Jessica C Cardenas; Charles E Wade; Bryan A Cotton; Mitchell J George; John B Holcomb; Martin A Schreiber; Nathan J White
Journal:  Shock       Date:  2019-03       Impact factor: 3.454

2.  The European guideline on management of major bleeding and coagulopathy following trauma: fifth edition.

Authors:  Donat R Spahn; Bertil Bouillon; Vladimir Cerny; Jacques Duranteau; Daniela Filipescu; Beverley J Hunt; Radko Komadina; Marc Maegele; Giuseppe Nardi; Louis Riddez; Charles-Marc Samama; Jean-Louis Vincent; Rolf Rossaint
Journal:  Crit Care       Date:  2019-03-27       Impact factor: 9.097

3.  Platelet-mimicking procoagulant nanoparticles augment hemostasis in animal models of bleeding.

Authors:  Ujjal Didar Singh Sekhon; Kelsey Swingle; Aditya Girish; Norman Luc; Maria de la Fuente; Jurgis Alvikas; Shannon Haldeman; Adnan Hassoune; Kaisal Shah; Youjoung Kim; Steven Eppell; Jeffrey Capadona; Andrew Shoffstall; Matthew D Neal; Wei Li; Marvin Nieman; Anirban Sen Gupta
Journal:  Sci Transl Med       Date:  2022-01-26       Impact factor: 19.319

Review 4.  Plasminogen activation in the musculoskeletal acute phase response: Injury, repair, and disease.

Authors:  Breanne H Y Gibson; Matthew T Duvernay; Stephanie N Moore-Lotridge; Matthew J Flick; Jonathan G Schoenecker
Journal:  Res Pract Thromb Haemost       Date:  2020-06-14

5.  Plasmin drives burn-induced systemic inflammatory response syndrome.

Authors:  Breanne H Y Gibson; Colby C Wollenman; Stephanie N Moore-Lotridge; Patrick R Keller; J Blair Summitt; Alexey R Revenko; Matthew J Flick; Timothy S Blackwell; Jonathan G Schoenecker
Journal:  JCI Insight       Date:  2021-12-08
  5 in total

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