Literature DB >> 28913856

Multiplate and TEG platelet mapping in a population of severely injured trauma patients.

M J George1, J Burchfield1, B MacFarlane1, Y-W W Wang1, J C Cardenas1, N J White2, B S Gill1, C E Wade1.   

Abstract

OBJECTIVES: The objectives of this study were to compare thromboelastography platelet mapping (TEG PM) with impedance aggregometry (Multiplate, MP) in a single trauma population and relate their results clinically.
BACKGROUND: Platelet function as measured by thromboelastography and impedance aggregometry demonstrates significant reductions that persist for days following traumatic injury. However, no study compares these devices and the correlation between them is not known.
METHODS: In level 1 trauma patients, TEG PM and MP were conducted at their initial presentation to the emergency department. Within-device repeatability and between-device association were determined using correlation analyses. Demographic variables, Injury Severity Score, blood product transfusion, laboratory test results and mortality rate were recorded.
RESULTS: Ninety-two patients were enrolled. Within-device repeatability was high for TEG PM and MP for arachidonic acid (AA) and adenosine diphosphate (ADP) activation pathways. When comparing TEG PM with MP, results correlated poorly in the ADP pathway (Spearman's rho = 0·11, P = 0·44) and moderately in the AA pathway (Spearman's rho = 0·56, P < 0·0001). TEG PM was predictive of blood product transfusion and correlated with increased base deficit, whereas MP was only predictive of mortality.
CONCLUSIONS: Intra-device variability was low for TEG PM and MP, but the two point-of-care devices measuring platelet function correlate poorly with each other in injured trauma patients. Each device also had different clinical associations.
© 2017 British Blood Transfusion Society.

Entities:  

Keywords:  impedance aggregometry; platelet function; thromboelastography; trauma

Mesh:

Year:  2017        PMID: 28913856      PMCID: PMC5854506          DOI: 10.1111/tme.12473

Source DB:  PubMed          Journal:  Transfus Med        ISSN: 0958-7578            Impact factor:   2.019


  19 in total

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2.  The platelet cytoskeleton regulates the aggregation-dependent synthesis of phosphatidylinositol 3,4-bisphosphate induced by thrombin.

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3.  Platelet function following trauma. A multiple electrode aggregometry study.

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4.  Early platelet dysfunction: an unrecognized role in the acute coagulopathy of trauma.

Authors:  Max V Wohlauer; Ernest E Moore; Scott Thomas; Angela Sauaia; Ed Evans; Jeffrey Harr; Christopher C Silliman; Victoria Ploplis; Francis J Castellino; Mark Walsh
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Authors:  R C Jacoby; J T Owings; J Holmes; F D Battistella; R C Gosselin; T G Paglieroni
Journal:  J Trauma       Date:  2001-10

6.  Platelets are dominant contributors to hypercoagulability after injury.

Authors:  Jeffrey N Harr; Ernest E Moore; Theresa L Chin; Arsen Ghasabyan; Eduardo Gonzalez; Max V Wohlauer; Anirban Banerjee; Christopher C Silliman; Angela Sauaia
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7.  Characterization of platelet dysfunction after trauma.

Authors:  Matthew E Kutcher; Brittney J Redick; Ryan C McCreery; Ian M Crane; Molly D Greenberg; Leslie M Cachola; Mary F Nelson; Mitchell Jay Cohen
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8.  Inhibition of platelet function is common following even minor injury.

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9.  Effects of antimycin A and 2-deoxyglucose on secretion in human platelets. Differential inhibition of the secretion of acid hydrolases and adenine nucleotides.

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Journal:  Biochem J       Date:  1979-08-15       Impact factor: 3.857

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Journal:  Thromb J       Date:  2007-02-20
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6.  Contractile forces in platelet aggregates under microfluidic shear gradients reflect platelet inhibition and bleeding risk.

Authors:  Lucas H Ting; Shirin Feghhi; Nikita Taparia; Annie O Smith; Ari Karchin; Esther Lim; Alex St John; Xu Wang; Tessa Rue; Nathan J White; Nathan J Sniadecki
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7.  TEG Platelet Mapping and Impedance Aggregometry to Predict Platelet Transfusion During Cardiopulmonary Bypass in Pediatric Patients.

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  10 in total

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