Literature DB >> 28872970

Interleukin-4 and Interleukin-10 Gene Polymorphisms in Patients with Inflammatory Bowel Disease.

Nasser Ebrahimi Daryani1, Amene Saghazadeh2,3, Shirin Moossavi4, Maryam Sadr2, Sepideh Shahkarami2,5, Samaneh Soltani2, Elham Farhadi6, Nima Rezaei7,8,9.   

Abstract

BACKGROUND: Changes in cytokine expression have been frequently found in patients with inflammatory bowel disease (IBD). Cytokine values outside the normal range may be somewhat related to common polymorphisms within cytokine genes.
OBJECTIVE: The present study was designed to investigate the possible association between polymorphisms within Interleukin IL-4 and IL-10 genes and susceptibility to and clinical features of IBD.
METHODS: The study population was composed of 140 healthy controls and 75 patients with IBD (40 patients with Crohn's disease (CD) and 35 patients with ulcerative colitis (UC)). Genotyping was performed using polymerase chain reaction with sequence-specific primers.
RESULTS: Higher frequencies for the C allele of IL-4-590 polymorphism (P < 0.0001; odds ratio [OR], 5.68; 95% confidence interval [95% CI], 3.28-9.83) and for the T allele of IL-4-1098 polymorphism (P = 0.016; OR, 1.83; 95% CI, 1.11-3.02) were observed in the whole group of IBD patients. The IL-4-590 C allele was also significantly overrepresented when IBD patients were subdivided into CD and UC (P < 0.0001; OR, 5.2-6.28). While the IL-4-1098 T allele was present at higher frequencies in patients with UC (P = 0.05; OR, 1.95), but not in CD (P = 0.09). Multiple pairwise comparisons indicated that genotypes of all polymorphisms investigated within IL-4 gene are correlated with IBD, CD, and UC. Haplotype analysis showed that the IL-4-1098/-590 TC haplotype might predispose individuals to IBD, CD, and UC whereas the IL-4-1098/-590 TT and GC haplotypes have a protective effect. On the contrary, neither allele nor genotype frequencies of IL-10 polymorphisms (IL-10-1082 A > G, IL-10-592 A > C, and IL-10-819 T > C) were associated with IBD, CD, or UC.
CONCLUSIONS: The present study suggests that IL-4 polymorphisms might play a role in susceptibility to IBD and its major subtypes in the Iranian population.

Entities:  

Keywords:  Crohn’s disease; IL-10; IL-4; Th2 cytokines; genetics; inflammatory bowel disease; polymorphisms; ulcerative colitis

Mesh:

Substances:

Year:  2017        PMID: 28872970     DOI: 10.1080/08820139.2017.1360343

Source DB:  PubMed          Journal:  Immunol Invest        ISSN: 0882-0139            Impact factor:   3.657


  6 in total

1.  Crescentic IgA nephropathy after administration of human monoclonal interleukin-12/23p40 antibody in a patient with Crohn's disease: a case report.

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Journal:  CEN Case Rep       Date:  2020-02-25

2.  Plasma Non-transferrin-Bound Iron Could Enter into Mice Duodenum and Negatively Affect Duodenal Defense Response to Virus and Immune Responses.

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Review 3.  Effector T Helper Cell Subsets in Inflammatory Bowel Diseases.

Authors:  Tanbeena Imam; Sungtae Park; Mark H Kaplan; Matthew R Olson
Journal:  Front Immunol       Date:  2018-06-01       Impact factor: 7.561

4.  miR-30c-5p Reduces Renal Ischemia-Reperfusion Involving Macrophage.

Authors:  Chengjun Zhang; Shengqiang Yu; Binyan Zheng; Dongfu Liu; Fengchun Wan; Yue Ma; Jiantao Wang; Zhenli Gao; Zhengfei Shan
Journal:  Med Sci Monit       Date:  2019-06-11

5.  IL-1β pre-stimulation enhances the therapeutic effects of endometrial regenerative cells on experimental colitis.

Authors:  Dingding Yu; Yiming Zhao; Hongda Wang; Dejun Kong; Wang Jin; Yonghao Hu; Yafei Qin; Baoren Zhang; Xiang Li; Jingpeng Hao; Guangming Li; Hao Wang
Journal:  Stem Cell Res Ther       Date:  2021-06-05       Impact factor: 6.832

6.  Barbaloin Attenuates Mucosal Damage in Experimental Models of Rat Colitis by Regulating Inflammation and the AMPK Signaling Pathway.

Authors:  Ling Gai; Likai Chu; Rui Xia; Qian Chen; Xingwei Sun
Journal:  Med Sci Monit       Date:  2019-12-27
  6 in total

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