Literature DB >> 28851046

Rise of PD-L1 expression during metastasis of colorectal cancer: Implications for immunotherapy.

Huan Bin Wang1, Han Yao1, Chu Shu Li1, Lun Xi Liang1, Yao Zhang1, Ying Xuan Chen1, Jing-Yuan Fang1, Jie Xu1.   

Abstract

OBJECTIVE: Programmed death-ligand 1 (PD-L1) expression in colorectal cancer (CRC) was implicated in predicting anti-PD-1/PD-L1 therapy efficacy. However, therapeutic response has also been found in patients without PD-L1 expression in the primary tumor. In the present study, we aimed to clarify the prevalence of PD-L1 in primary and metastatic CRC.
METHODS: The expression of PD-L1 was determined by immunohistochemistry in matched primary and metastatic CRC.
RESULTS: PD-L1 expression was significantly more prevalent in metastatic CRCs than in primary tumors, and the expression of PD-L1 in primary CRC may not represent the tumors that spread to distant organs. Positive expression of PD-L1 was found in 81.8% of metastatic CRC, being significantly more prevalent than in primary CRC (40.9%; P = 0.012, Fisher's exact test). While comparing the primary and metastatic lesions of the same patients, we found that PD-L1 expression frequently increased during the metastatic process. However, PD-L1 expression was rarely decreased in metastatic lesions. Intratumoral heterogeneity expression of PD-L1 was found in both metastatic CRC (22.2%) and primary CRCs (33.3%). PD-L1 was prevalently expressed in metastatic CRC, and increased PD-L1 expression was frequently found in metastatic CRC as compared to primary tumors.
CONCLUSION: PD-L1 expression in metastatic CRC should be considered as an independent factor while evaluating the suitability of patients for immunotherapy.
© 2017 Chinese Medical Association Shanghai Branch, Chinese Society of Gastroenterology, Renji Hospital Affiliated to Shanghai Jiaotong University School of Medicine and John Wiley & Sons Australia, Ltd.

Entities:  

Keywords:  PD-L1; colorectal neoplasms; immune checkpoint blockade; neoplasm metastasis

Mesh:

Substances:

Year:  2017        PMID: 28851046     DOI: 10.1111/1751-2980.12538

Source DB:  PubMed          Journal:  J Dig Dis        ISSN: 1751-2972            Impact factor:   2.325


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