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Literature DB >> 28839090

Reply to "Early Stochastic Dynamics in Human Cytomegalovirus Infection".

Soren Gantt^1,2, Bryan T Mayer², Joshua T Schiffer^2,3.

Abstract

Entities: Disease

Mesh：

Year: 2017 PMID： 28839090 PMCID： PMC5571264 DOI： 10.1128/JVI.01006-17

Source DB: PubMed Journal: J Virol ISSN： 0022-538X Impact factor: 5.103

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8 in total

1. Early Stochastic Dynamics in Human Cytomegalovirus Infection.

Authors: Dominik Wodarz; Ignacio Rodriguez-Brenes
Journal: J Virol Date: 2017-08-24 Impact factor: 5.103

2. Human Cytomegalovirus Tegument Protein UL82 Inhibits STING-Mediated Signaling to Evade Antiviral Immunity.

Authors: Yu-Zhi Fu; Shan Su; Yi-Qun Gao; Pei-Pei Wang; Zhe-Fu Huang; Ming-Ming Hu; Wei-Wei Luo; Shu Li; Min-Hua Luo; Yan-Yi Wang; Hong-Bing Shu
Journal: Cell Host Microbe Date: 2017-01-26 Impact factor: 21.023

3. Transient Oral Human Cytomegalovirus Infections Indicate Inefficient Viral Spread from Very Few Initially Infected Cells.

Authors: Bryan T Mayer; Elizabeth M Krantz; David Swan; James Ferrenberg; Karen Simmons; Stacy Selke; Meei-Li Huang; Corey Casper; Lawrence Corey; Anna Wald; Joshua T Schiffer; Soren Gantt
Journal: J Virol Date: 2017-05-26 Impact factor: 5.103

Reply to "Early Stochastic Dynamics in Human Cytomegalovirus Infection".

1. Early Stochastic Dynamics in Human Cytomegalovirus Infection.

2. Human Cytomegalovirus Tegument Protein UL82 Inhibits STING-Mediated Signaling to Evade Antiviral Immunity.

3. Transient Oral Human Cytomegalovirus Infections Indicate Inefficient Viral Spread from Very Few Initially Infected Cells.

4. Dynamics of Persistent Oral Cytomegalovirus Shedding During Primary Infection in Ugandan Infants.

5. Human Cytomegalovirus pTRS1 and pIRS1 Antagonize Protein Kinase R To Facilitate Virus Replication.

6. A cytomegaloviral protein reveals a dual role for STAT2 in IFN-{gamma} signaling and antiviral responses.

7. Type 1 Interferons and NK Cells Limit Murine Cytomegalovirus Escape from the Lymph Node Subcapsular Sinus.

8. Complex Dynamics of Virus Spread from Low Infection Multiplicities: Implications for the Spread of Oncolytic Viruses.