| Literature DB >> 28816705 |
Austin T Robinson1, Ibra S Fancher1,1, Abeer M Mahmoud1,1,1, Shane A Phillips1,1,1.
Abstract
Endothelium-dependent vasodilation is reduced after acute exercise or after high intraluminal pressure in isolated arterioles from sedentary adults but not in arterioles from regular exercisers. The preserved vasodilation in arterioles from exercisers is hydrogen peroxide (H2O2) dependent, whereas resting dilation is nitric oxide (NO) dependent. We hypothesize chronic exercise elicits adaptations allowing for maintained vasodilation when NO bioavailability is reduced.Entities:
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Year: 2018 PMID: 28816705 PMCID: PMC5735031 DOI: 10.1249/JES.0000000000000130
Source DB: PubMed Journal: Exerc Sport Sci Rev ISSN: 0091-6331 Impact factor: 6.230