Literature DB >> 28812426

The expression of ETAR in liver cirrhosis and liver cancer.

Juhong Deng1,2, Yu Huang3, Ran Tao1, Xiangxue Fan4, Hongyue Zhang1, Hongyan Kong1, Qiqing Song1, Jiaquan Huang1.   

Abstract

BACKGROUND: To investigate the expression of endothelin receptors in liver diseases and discuss its role in the process of liver cirrhosis and liver cancer. RESEARCH DESIGN AND METHODS: We examined the expressions of ETAR, ETBR and α-SMA in tissue samples using western blotting analysis. Furthermore, immunofluorescence was used to locate ETAR expression in hepatic stellate cells (HSCs) and hepatic sinusoidal endothelial cells (HSECs), we calculated the percentage of positive cells and then analyzed its relation with clinical indexes.
RESULTS: According to the western blotting analysis, the expression of ETAR was high in hepatic hemangioma and liver cancer tissues and ETBR was highly expressed in cirrhosis tissues. The immunofluorescence results demonstrated that the expression of ETAR was elevated in hepatic hemangioma and liver cancer tissues. Moreover, ETAR expression was found in both HSCs and HSECs. Finally, the statistical analysis revealed that the number of positive ETAR cells was correlated with the clinical index platelets (PLT), alanine transaminase (ALT) and diameter of portal vein.
CONCLUSION: Endothelin receptors express differently in liver cirrhosis and liver cancer tissues and play a role in hepatic diseases by affecting HSCs and HSECs.

Entities:  

Keywords:  ETAR; HSCs and HSECs; liver cancer; liver cirrhosis

Mesh:

Substances:

Year:  2017        PMID: 28812426      PMCID: PMC5663415          DOI: 10.1080/15384047.2017.1360451

Source DB:  PubMed          Journal:  Cancer Biol Ther        ISSN: 1538-4047            Impact factor:   4.742


  29 in total

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10.  Global, regional, and national age-sex specific all-cause and cause-specific mortality for 240 causes of death, 1990-2013: a systematic analysis for the Global Burden of Disease Study 2013.

Authors: 
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  1 in total

1.  Endothelin receptors promote schistosomiasis-induced hepatic fibrosis via splenic B cells.

Authors:  Hongyan Kong; Jinan He; Shusen Guo; Qiqin Song; Dandan Xiang; Ran Tao; Haijing Yu; Guang Chen; Zhiyong Huang; Qin Ning; Jiaquan Huang
Journal:  PLoS Pathog       Date:  2020-10-19       Impact factor: 6.823

  1 in total

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