Literature DB >> 28793999

Association of QT-Prolonging Medication Use in CKD with Electrocardiographic Manifestations.

Soren Snitker1, Rebecca M Doerfler1, Elsayed Z Soliman1, Rajat Deo1, Wendy L St Peter1, Susan Kramlik1, Michael J Fischer1, Sankar Navaneethan1, Patrice Delafontaine1, Bernard G Jaar1, Akinlolu Ojo1, Gail K Makos1, Anne Slaven1, Matthew R Weir1, Min Zhan1, Jeffrey C Fink2.   

Abstract

BACKGROUND AND OBJECTIVES: Several drugs used in CKD can prolong electrocardiographic conduction. We examined the use of electrocardiogram QT-prolonging medications in predialysis CKD and their association with QT duration. DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: In total, 3252 Chronic Renal Insufficiency Cohort participants with at least one study electrocardiogram between 2003 and 2011 were included. QT-prolonging medications used in 100 or more visits (n=16,451 visits) along with diuretics and proton pump inhibitors, given their potential for electrolyte disturbances, were examined for QT interval prolongation.
RESULTS: Mean QT interval corrected for heart rate was at 414±21 (±SD) milliseconds and prolonged (≥450 milliseconds) in 4.6% of electrocardiograms. QT interval corrected for heart rate was inversely related to serum potassium and calcium. Medications classified as QT prolonging were taken at 76% of visits, with two or more of these taken at 33% of visits. Of 30 medications examined, eight were associated with statistically significant QT interval corrected for heart rate prolongation after adjustment for comorbidities, potassium, and calcium, including amiodarone (+10±2 milliseconds), metolazone (+7±2 milliseconds), fluoxetine (+4±1 milliseconds), citalopram (+4±1 milliseconds), hydroxyzine (+4±1 milliseconds), escitalopram (+3±2 milliseconds), venlafaxine (+3±1 milliseconds), and furosemide (+3±0 milliseconds). Potassium-depleting diuretics were associated with minimal decrements in potassium (between 0.1 and 0.3 mEq/L) and smaller changes in calcium. Diuretics associated with a change in QT interval corrected for heart rate before adjustment for potassium and calcium were metolazone (+8±3 milliseconds), furosemide (+4±1 milliseconds), and spironolactone (-3±3 milliseconds). Most of the QT prolongation associated with metolazone and furosemide, but not spironolactone, remained after adjustment for potassium and calcium. Proton pump inhibitors were not associated with QT prolongation.
CONCLUSIONS: Use of medications associated with QT prolongation is common in CKD; the safety implications of these findings should be considered in these high-risk patients. PODCAST: This article contains a podcast at https://www.asn-online.org/media/podcast/CJASN/2017_08_09_CJASNPodcast_17_09_b.mp3.
Copyright © 2017 by the American Society of Nephrology.

Entities:  

Keywords:  Amiodarone; Arrhythmias, Cardiac; Brugada Syndrome; Cardiac Conduction Defect; Citalopram; Diuretics; Electrocardiography; Electrolytes; Fluoxetine; Furosemide; Heart Conduction System; Humans; Hydroxyzine; Metolazone; Potassium; Proton Pump Inhibitors; Renal Dialysis; Renal Insufficiency, Chronic; Sodium Chloride Symporter Inhibitors; Spironolactone; Venlafaxine Hydrochloride

Mesh:

Substances:

Year:  2017        PMID: 28793999      PMCID: PMC5586585          DOI: 10.2215/CJN.12991216

Source DB:  PubMed          Journal:  Clin J Am Soc Nephrol        ISSN: 1555-9041            Impact factor:   8.237


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