Literature DB >> 28731263

Commercial strain-derived clinical Saccharomyces cerevisiae can evolve new phenotypes without higher pathogenicity.

Walter P Pfliegler1,2, Enikő Boros1, Kitti Pázmándi3, Ágnes Jakab1, Imre Zsuga1, Renátó Kovács4,5, Edit Urbán6, Zsuzsa Antunovics7, Attila Bácsi3, Matthias Sipiczki7, László Majoros4, István Pócsi1.   

Abstract

SCOPE: Saccharomyces cerevisiae is one of the most important microbes in food industry, but there is growing evidence on its potential pathogenicity as well. Its status as a member of human mycobiome is still not fully understood. METHODS AND
RESULTS: In this study, we characterize clinical S. cerevisiae isolates from Hungarian hospitals along with commercial baking and probiotic strains, and determine their phenotypic parameters, virulence factors, interactions with human macrophages, and pathogenicity. Four of the clinical isolates could be traced back to commercial strains based on genetic fingerprinting. Our observations indicate that the commercial-derived clinical isolates have evolved new phenotypes and show similar, or in two cases, significantly decreased pathogenicity. Furthermore, immunological experiments revealed that the variability in human primary macrophage activation after coincubation with yeasts is largely donor and not isolate dependent.
CONCLUSION: Isolates in this study offer an interesting insight into the potential microevolution of probiotic and food strains in human hosts. These commensal yeasts display various changes in their phenotypes, indicating that the colonization of the host does not necessarily impose a selective pressure toward higher virulence/pathogenicity.
© 2017 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim.

Entities:  

Keywords:  Baking yeast; Commensal; Microevolution; Mycobiom; Probiotic

Mesh:

Substances:

Year:  2017        PMID: 28731263     DOI: 10.1002/mnfr.201601099

Source DB:  PubMed          Journal:  Mol Nutr Food Res        ISSN: 1613-4125            Impact factor:   5.914


  2 in total

1.  Heme Oxygenase-1 (HMX1) Loss of Function Increases the In-Host Fitness of the Saccharomyces 'boulardii' Probiotic Yeast in a Mouse Fungemia Model.

Authors:  Alexandra Imre; Renátó Kovács; Zoltán Tóth; László Majoros; Zsigmond Benkő; Walter P Pfliegler; István Pócsi
Journal:  J Fungi (Basel)       Date:  2022-05-18

2.  Uric acid levels are independent of anti-Saccharomyces cerevisiae antibodies (ASCA) in Crohn's disease: A reappraisal of the role of S. cerevisiae in this setting.

Authors:  B Sendid; S Jawhara; H Sarter; P Maboudou; C Thierny; C Gower-Rousseau; J F Colombel; D Poulain
Journal:  Virulence       Date:  2018       Impact factor: 5.882

  2 in total

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