Literature DB >> 28714083

Acute liver failure with amiodarone infusion: A case report and systematic review.

P Jaiswal1, B M Attar2, J E Yap2, K Devani3, R Jaiswal4, Y Wang1, R Szynkarek5, D Patel6, M Demetria2.   

Abstract

WHAT IS KNOWN AND
OBJECTIVE: Amiodarone, a commonly used class III antiarrhythmic agent notable for a relatively long half-life of up to 6 months and its pronounced adverse effect profile, is used for both acute and chronic management of cardiac arrhythmias. Chronic use of amiodarone has been associated with asymptomatic hepatotoxicity; however, acute toxicity is thought to be uncommon. There are only six reported cases of acute liver failure (ALF) secondary to amiodarone. In all these cases the outcome of death during the same hospitalization resulted. We aimed to report the only case of acute liver failure secondary to amiodarone infusion in the existing literature where the patient survived. CASE
SUMMARY: A 79-year-old woman admitted with atrial flutter was being treated with intravenous (IV) amiodarone when she abruptly developed coagulopathy, altered mental status and liver enzyme derangement. She was diagnosed with acute liver failure (ALF) secondary to an amiodarone adverse drug reaction, with a calculated score of seven on the Naranjo adverse drug reaction probability scale. Amiodarone was immediately withheld, and N-acetylcysteine (NAC) was initiated. Clinical improvement was seen within 48 hours of holding the drug and within 24 hours of initiating NAC. On post-hospital follow-up visit she was reported to have complete recovery. WHAT IS NEW AND
CONCLUSION: This report emphasizes the importance of monitoring liver enzymes and mental status while a patient is being administered IV amiodarone. N-acetylcysteine administration may have possibly contributed to the early and successful recovery from ALF in our patient. To date, she is the only patient in the existing literature who has been reported to survive ALF secondary to amiodarone administration.
© 2017 John Wiley & Sons Ltd.

Entities:  

Keywords:  N-acetylcysteine; acute hepatic failure; acute liver failure; amiodarone; systematic review; treatment

Mesh:

Substances:

Year:  2017        PMID: 28714083     DOI: 10.1111/jcpt.12594

Source DB:  PubMed          Journal:  J Clin Pharm Ther        ISSN: 0269-4727            Impact factor:   2.512


  4 in total

1.  The protective potential of alpha lipoic acid on amiodarone-induced pulmonary fibrosis and hepatic injury in rats.

Authors:  Ghadha Ibrahim Fouad; Mohamed R Mousa
Journal:  Mol Cell Biochem       Date:  2021-05-10       Impact factor: 3.396

2.  Emerging club drugs: 5-(2-aminopropyl)benzofuran (5-APB) is more toxic than its isomer 6-(2-aminopropyl)benzofuran (6-APB) in hepatocyte cellular models.

Authors:  Rita Roque Bravo; Helena Carmo; João Pedro Silva; Maria João Valente; Félix Carvalho; Maria de Lourdes Bastos; Diana Dias da Silva
Journal:  Arch Toxicol       Date:  2019-12-14       Impact factor: 5.153

3.  Altered Mental Status and Hyponatremia After 20 Hours of Amiodarone Therapy.

Authors:  Andrea Qi; John C Moscona; Justin Reed; Thierry H Le Jemtel
Journal:  Tex Heart Inst J       Date:  2020-06-01

4.  Concomitant Acute Hepatic Failure and Renal Failure Induced by Intravenous Amiodarone: A Case Report and Literature Review.

Authors:  Mujtaba Mohamed; Alsadiq Al-Hillan; Marcus Flores; Christian Kaunzinger; Arman Mushtaq; Arif Asif; Mohammad Hossain
Journal:  Gastroenterology Res       Date:  2020-02-01
  4 in total

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