Literature DB >> 28707191

Clostridium Difficile, Colitis, and Colonoscopy: Pediatric Perspective.

Randolph McConnie1,2, Arthur Kastl3.   

Abstract

PURPOSE OF THE REVIEW: Review tests available for detection of Clostridium difficile (C. Diff) induced disease, including when such tests should be done in children and how they should be interpreted. RECENT
FINDINGS: Multiple tests are available for detecting disease due to C. diff. These include colonoscopy and stool analysis. Colonoscopy with biopsy is the most sensitive test for detecting the presence of colitis. The toxins produced by the C. diff. (toxin A, toxin B, and binary toxin) are the agents that cause injury and disease. Only toxin producing C. diff. Strains will cause disease. Binary toxin by itself is not thought to produce disease. Binary toxin causes disease in humans when present with toxin A and B producing bacteria, and has been implicated with fulminant life threatening disease. Stool analyses vary in sensitivity and specificity depending on the assay used. The presence of toxin producing strains of C diff. in the stool does not equate with disease. The presence of a toxin-producing bacteria or toxins (A or B) only equates with disease if diarrhea or a diseased colon (toxic megacolon, ileus, and sepsis) is present. Nucleic acid amplification testing (NAAT), when used in the stool from patients with diarrhea, appears to be the most efficient study to detect the gene that encodes for toxin A and B and thus to diagnose C. diff.-induced disease. Infants have a high carriage rate of C. diff. and are believed not to develop disease from it or its toxins. Infants should not be tested for C. difficile. The NAAT is most specific when done on patients with diarrhea with liquid stools. Testing for C. difficile should only be done on patients with diarrhea. One can assume that a patient who has no diarrhea and is not ill does not have C. diff.-induced disease. Treatment should be limited to patients with diarrhea who test positive for C. diff. toxin (A or B) or toxin-producing bacteria. Direct testing for binary toxin is not commercially available. Binary toxin is only thought to cause disease in humans when C. diff. toxin (A and B)-producing bacteria are present.

Entities:  

Keywords:  Binary toxin; C. diff.; C. diff. toxin; Children; Clostridium difficile; Colitis; Colonoscopy; NAP1; Pediatric; Pseudomembranous colitis; Toxin A; Toxin B

Mesh:

Substances:

Year:  2017        PMID: 28707191     DOI: 10.1007/s11894-017-0571-z

Source DB:  PubMed          Journal:  Curr Gastroenterol Rep        ISSN: 1522-8037


  26 in total

1.  Use of Acid Suppression Medication is Associated With Risk for C. difficile Infection in Infants and Children: A Population-based Study.

Authors:  Daniel E Freedberg; Esi S Lamousé-Smith; Jenifer R Lightdale; Zhezhen Jin; Yu-Xiao Yang; Julian A Abrams
Journal:  Clin Infect Dis       Date:  2015-06-09       Impact factor: 9.079

2.  NAP1 strain type predicts outcomes from Clostridium difficile infection.

Authors:  Isaac See; Yi Mu; Jessica Cohen; Zintars G Beldavs; Lisa G Winston; Ghinwa Dumyati; Stacy Holzbauer; John Dunn; Monica M Farley; Carol Lyons; Helen Johnston; Erin Phipps; Rebecca Perlmutter; Lydia Anderson; Dale N Gerding; Fernanda C Lessa
Journal:  Clin Infect Dis       Date:  2014-03-05       Impact factor: 9.079

Review 3.  Detecting and treating Clostridium difficile infections in patients with inflammatory bowel disease.

Authors:  Ashwin N Ananthakrishnan
Journal:  Gastroenterol Clin North Am       Date:  2012-02-14       Impact factor: 3.806

4.  Real-time polymerase chain reaction detection of asymptomatic Clostridium difficile colonization and rising C. difficile-associated disease rates.

Authors:  Hoonmo L Koo; John N Van; Meina Zhao; Xunyan Ye; Paula A Revell; Zhi-Dong Jiang; Carolyn Z Grimes; Diana C Koo; Todd Lasco; Claudia A Kozinetz; Kevin W Garey; Herbert L DuPont
Journal:  Infect Control Hosp Epidemiol       Date:  2014-04-22       Impact factor: 3.254

Review 5.  Diagnosis of Clostridium difficile infection: an ongoing conundrum for clinicians and for clinical laboratories.

Authors:  Carey-Ann D Burnham; Karen C Carroll
Journal:  Clin Microbiol Rev       Date:  2013-07       Impact factor: 26.132

6.  Clostridium difficile carriage in healthy infants in the community: a potential reservoir for pathogenic strains.

Authors:  Clotilde Rousseau; Isabelle Poilane; Loic De Pontual; Anne-Claire Maherault; Alban Le Monnier; Anne Collignon
Journal:  Clin Infect Dis       Date:  2012-07-25       Impact factor: 9.079

7.  Clinical Significance of Clostridium difficile in Children Less Than 2 Years Old: A Case-Control Study.

Authors:  Marcela González-Del Vecchio; Ana Álvarez-Uria; Mercedes Marin; Luis Alcalá; Adoración Martín; Pedro Montilla; Emilio Bouza
Journal:  Pediatr Infect Dis J       Date:  2016-03       Impact factor: 2.129

8.  Antibiotic-associated pseudomembranous colitis due to toxin-producing clostridia.

Authors:  J G Bartlett; T W Chang; M Gurwith; S L Gorbach; A B Onderdonk
Journal:  N Engl J Med       Date:  1978-03-09       Impact factor: 91.245

9.  Impact of Clostridium difficile on inflammatory bowel disease.

Authors:  Mazen Issa; Aravind Vijayapal; Mary Beth Graham; Dawn B Beaulieu; Mary F Otterson; Sarah Lundeen; Susan Skaros; Lydia R Weber; Richard A Komorowski; Josh F Knox; Jeanne Emmons; Jasmohan S Bajaj; David G Binion
Journal:  Clin Gastroenterol Hepatol       Date:  2007-03       Impact factor: 11.382

10.  Immunoglobulin and non-immunoglobulin components of human milk inhibit Clostridium difficile toxin A-receptor binding.

Authors:  R D Rolfe; W Song
Journal:  J Med Microbiol       Date:  1995-01       Impact factor: 2.472

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