Literature DB >> 28679849

Mendelian Genes and Risk of Intracerebral Hemorrhage and Small-Vessel Ischemic Stroke in Sporadic Cases.

Michael Chong1, Martin O'Donnell1, Vincent Thijs1, Antonio Dans1, Patricio López-Jaramillo1, Diego Gómez-Arbeláez1, Charles Mondo1, Anna Czlonkowska1, Marta Skowronska1, Shahram Oveisgharan1, Salim Yusuf1, Guillaume Paré2.   

Abstract

BACKGROUND AND
PURPOSE: Mendelian strokes are rare genetic disorders characterized by early-onset small-vessel stroke. Although extensively studied among families with syndromic features, whether these genes affect risk among sporadic cases is unknown.
METHODS: We sequenced 8 genes responsible for Mendelian stroke in a case-control study of sporadic stroke cases (≤70 years). Participants included 1251 primary stroke cases of small-vessel pathology (637 intracerebral hemorrhage and 614 small-vessel ischemic stroke cases) and 1716 controls from the INTERSTROKE study (Study of the Importance of Conventional and Emerging Risk Factors of Stroke in Different Regions and Ethnic Groups of the World).
RESULTS: Overall, the prevalence of canonical disease-causing mutations was 0.56% in cases and 0.23% in controls (odds ratio=1.89; 95% confidence interval, 0.54-7.57; P=0.33). CADASIL (Cerebral Autosomal Dominant Arteriopathies with Subcortical Infarcts and Leukoencephalopathies) mutations were more frequent among cases (0.48%) than controls (0.23%) but were not significantly associated with stroke risk (odds ratio=2.03; 95% confidence interval, 0.58-8.02; P=0.27). Next, we included all rare nonsynonymous mutations to investigate whether other types of mutations may contribute to stroke risk. Overall, 13.5% of cases and 14.2% of controls were carriers of at least one rare nonsynonymous mutation among the 8 Mendelian stroke genes. Mutation carriers were not at elevated risk of stroke (odds ratio=0.93; 95% confidence interval, 0.75-1.16; P=0.55).
CONCLUSIONS: In the absence of syndromic features and family history of stroke, screening for Mendelian mutations among small-vessel stroke patients is unlikely to have high diagnostic utility.
© 2017 American Heart Association, Inc.

Entities:  

Keywords:  CADASIL; case–control studies; genetics; prevalence; stroke

Mesh:

Year:  2017        PMID: 28679849     DOI: 10.1161/STROKEAHA.117.017322

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  7 in total

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6.  Rare variants of the 3'-5' DNA exonuclease TREX1 in early onset small vessel stroke.

Authors:  Hugh S Markus; Cathie Sudlow; David P J Hunt; Sarah McGlasson; Kristiina Rannikmäe; Steven Bevan; Clare Logan; Louise S Bicknell; Alexa Jury; Andrew P Jackson
Journal:  Wellcome Open Res       Date:  2017-11-02

7.  How common are single gene mutations as a cause for lacunar stroke? A targeted gene panel study.

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  7 in total

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