Literature DB >> 28671135

Fifteen Years of Clinical Trials in Huntington's Disease: A Very Low Clinical Drug Development Success Rate.

André M Travessa1, Filipe B Rodrigues1,2,3, Tiago A Mestre4, Joaquim J Ferreira1,2,5.   

Abstract

BACKGROUND: Drug development in Huntington's disease (HD) is particularly challenging, and only two compounds are approved by the FDA. It is therefore essential to appraise drug development programs in order to understand the reasons for their failure during the early stages of development.
OBJECTIVES: To describe the landscape of HD therapeutic development and critically explore the causes of compound attrition in the different stages of drug development, from phase 1 to phase 4.
METHODS: All HD clinical trials registered in the WHO International Clinical Trials Search Portal, from inception to May 2017, were analyzed. Two independent authors selected and extracted data. Success rate in a trial phase was calculated as the number of compounds that progressed to the next trial phase divided by the number of compounds in that phase. The overall success rate was calculated as the ratio between the number of compounds that receive regulatory approval and the total number of compounds.
RESULTS: Ninety-nine trials assessing 41 compounds and eleven non-pharmacological interventions (devices and cell therapies) were identified. Twenty-four (24.2%) were phase 1 trials, 46 (46.5%) phase 2, 20 (20.2%) phase 3, and two (2.0%) phase 4. Sixty trials (60.6%) received industry sponsorship. The most frequently studied compounds were creatine, latrepirdine and pridopidine. The mean number of participants enrolled was 92.0 and the length of treatment was 262.9 days, and both increased from phase 1 to phase 3 trials. The success rate was 25.0% from phase 1 to phase 2, 19.4% from phase 2 to phase 3, and 14.3% from phase 3 to approval. The overall success rate was 3.5%.
CONCLUSIONS: Although HD is a rare condition, 99 HD trials were identified in a comprehensive clinical trial registry. We found a low success rate at earlier phases of drug-development and a very low trial success rate at later phases. There is a significant gap between drug discovery and development success rates that warrants careful appraisal and improvement.

Entities:  

Keywords:  Clinical development; Huntington disease; clinical trials; medicines

Mesh:

Year:  2017        PMID: 28671135     DOI: 10.3233/JHD-170245

Source DB:  PubMed          Journal:  J Huntingtons Dis        ISSN: 1879-6397


  22 in total

Review 1.  Proteostasis in Huntington's disease: disease mechanisms and therapeutic opportunities.

Authors:  Rachel J Harding; Yu-Feng Tong
Journal:  Acta Pharmacol Sin       Date:  2018-04-05       Impact factor: 6.150

Review 2.  Therapeutic approaches to Huntington disease: from the bench to the clinic.

Authors:  Nicholas S Caron; E Ray Dorsey; Michael R Hayden
Journal:  Nat Rev Drug Discov       Date:  2018-09-21       Impact factor: 84.694

Review 3.  Therapeutic Update on Huntington's Disease: Symptomatic Treatments and Emerging Disease-Modifying Therapies.

Authors:  Deepa Dash; Tiago A Mestre
Journal:  Neurotherapeutics       Date:  2020-10       Impact factor: 7.620

4.  Agreement between clinician-rated versus patient-reported outcomes in Huntington disease.

Authors:  Noelle E Carlozzi; Nicholas R Boileau; Joel S Perlmutter; Kelvin L Chou; Julie C Stout; Jane S Paulsen; Michael K McCormack; David Cella; Martha A Nance; Jin-Shei Lai; Praveen Dayalu
Journal:  J Neurol       Date:  2018-04-23       Impact factor: 4.849

5.  Drp1/Fis1-mediated mitochondrial fragmentation leads to lysosomal dysfunction in cardiac models of Huntington's disease.

Authors:  A U Joshi; A E Ebert; B Haileselassie; D Mochly-Rosen
Journal:  J Mol Cell Cardiol       Date:  2018-12-11       Impact factor: 5.000

6.  The risks of converting post-hoc findings into primary outcomes in subsequent trials.

Authors:  Filipe B Rodrigues; Joaquim J Ferreira
Journal:  Ann Transl Med       Date:  2019-12

Review 7.  Recent Advances in the Treatment of Huntington's Disease: Targeting DNA and RNA.

Authors:  Kathleen M Shannon
Journal:  CNS Drugs       Date:  2020-03       Impact factor: 5.749

Review 8.  Translation of MicroRNA-Based Huntingtin-Lowering Therapies from Preclinical Studies to the Clinic.

Authors:  Jana Miniarikova; Melvin M Evers; Pavlina Konstantinova
Journal:  Mol Ther       Date:  2018-02-08       Impact factor: 11.454

9.  Mutant huntingtin and neurofilament light have distinct longitudinal dynamics in Huntington's disease.

Authors:  Filipe B Rodrigues; Lauren M Byrne; Rosanna Tortelli; Eileanoir B Johnson; Peter A Wijeratne; Marzena Arridge; Enrico De Vita; Naghmeh Ghazaleh; Richard Houghton; Hannah Furby; Daniel C Alexander; Sarah J Tabrizi; Scott Schobel; Rachael I Scahill; Amanda Heslegrave; Henrik Zetterberg; Edward J Wild
Journal:  Sci Transl Med       Date:  2020-12-16       Impact factor: 17.956

10.  Risks and benefits of unapproved disease-modifying treatments for neurodegenerative disease.

Authors:  Aden C Feustel; Amanda MacPherson; Dean A Fergusson; Karl Kieburtz; Jonathan Kimmelman
Journal:  Neurology       Date:  2019-12-02       Impact factor: 9.910

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