Literature DB >> 28666582

Clonidine restores vascular endothelial growth factor expression and improves tissue repair following severe trauma.

Tyler J Loftus1, Andrew J Thomson2, Kolenkode B Kannan3, Ines G Alamo4, Jessica K Millar5, Jessica M Plazas6, Elizabeth E Whitley7, Philip A Efron8, Alicia M Mohr9.   

Abstract

BACKGROUND: We hypothesized that clonidine and propranolol would increase VEGF and VEGF-receptor expression and promote lung healing following severe trauma and chronic stress.
METHODS: Sprague-Dawley rats were subjected to lung contusion (LC), lung contusion/hemorrhagic shock (LCHS), or lung contusion/hemorrhagic shock/daily restraint stress (LCHS/CS). Clonidine and propranolol were administered daily. On day seven, lung VEGF, VEGFR-1, VEGFR-2, and HMGB1 were assessed by PCR. Lung injury was assessed by light microscopy (*p < 0.05).
RESULTS: Clonidine increased VEGF expression following LCHS (43%*) and LCHS/CS (46%*). Clonidine increased VEGFR-1 and R-2 expression following LCHS/CS (203%* and 47%*, respectively). Clonidine decreased HMGB1 and TNF-alpha expression following LCHS/CS (22%* and 58%*, respectively.) Clonidine decreased inflammatory cell infiltration and total Lung Injury Score following LCHS/CS. Propranolol minimally affected VEGF and did not improve lung healing.
CONCLUSIONS: Clonidine increased VEGF and VEGF-receptor expression, decreased HMGB1 expression, decreased lung inflammation, and improved lung tissue repair.
Copyright © 2017 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Lung contusion; Stress; Tissue repair; Trauma; Vascular endothelial growth factor

Mesh:

Substances:

Year:  2017        PMID: 28666582      PMCID: PMC5603405          DOI: 10.1016/j.amjsurg.2017.06.019

Source DB:  PubMed          Journal:  Am J Surg        ISSN: 0002-9610            Impact factor:   2.565


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