Literature DB >> 28605468

Attenuation of Insulin Action by an Allosteric Insulin Receptor Antibody in Healthy Volunteers.

Kirk W Johnson1, Ann Neale1, Allan Gordon1, Julie Roessig1, Padma Bezwada1, Sabine Vukelich1, Ira Goldfine1, Paul Rubin1.   

Abstract

Background: XOMA 358 (X358) is a fully human monoclonal antibody to the insulin receptor that acts as a negative allosteric modulator of insulin signaling. It is being developed as a novel treatment of hyperinsulinemic hypoglycemia. This report describes pharmacokinetic (PK) and pharmacodynamic (PD) data from a first-in-human clinical trial.
Methods: A double-blind, placebo-controlled, single-ascending-dose study was performed with 29 healthy adult males randomized to intravenous infusion of placebo or X358 at 0.1-, 0.3-, 1-, 3-, 6-, or 9-mg/kg dose levels. The primary objective was to assess safety and tolerability, and secondary objectives included PK and PD analyses. A short insulin tolerance test (ITT) was implemented in the 3- to 9-mg/kg dose cohorts at baseline and postinfusion.
Results: There were no deaths, serious adverse events (AEs), or subject discontinuations due to AEs. There were no clinically meaningful safety findings. X358 exhibited dose-proportional PK with a half-life of 21 days. Dose-dependent elevations of circulating insulin levels, likely related to reduced insulin clearance via monoclonal antibody action at receptors, represented a sensitive biomarker of X358 exposure. X358-dependent increases in postprandial glucose levels and fasting homeostatic model assessment of insulin resistance values were observed and persisted for at least 1 week at the higher dose levels. In all the ITT cohorts, the slope for glucose lowering was substantially attenuated after X358 infusion of a similar magnitude, but with increasing duration with rising dose level.
Conclusion: Single X358 infusions were well tolerated and resulted in a dose-dependent reduction in insulin sensitivity. Clinical development of X358 in hyperinsulinemic, hypoglycemic conditions is proceeding.
Copyright © 2017 Endocrine Society

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Year:  2017        PMID: 28605468     DOI: 10.1210/jc.2017-00822

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  5 in total

1.  A unique allosteric insulin receptor monoclonal antibody that prevents hypoglycemia in the SUR-1-/- mouse model of KATP hyperinsulinism.

Authors:  Puja Patel; Lawrenshey Charles; John Corbin; Ira D Goldfine; Kirk Johnson; Paul Rubin; Diva D De León
Journal:  MAbs       Date:  2018-06-01       Impact factor: 5.857

2.  A Randomized, Placebo-Controlled Double-Blind Trial of a Closed-Loop Glucagon System for Postbariatric Hypoglycemia.

Authors:  Christopher M Mulla; Stamatina Zavitsanou; Alejandro Jose Laguna Sanz; David Pober; Lauren Richardson; Pamela Walcott; Ipsa Arora; Brett Newswanger; Martin J Cummins; Steve J Prestrelski; Francis J Doyle; Eyal Dassau; Mary Elizabeth Patti
Journal:  J Clin Endocrinol Metab       Date:  2020-04-01       Impact factor: 5.958

3.  Evaluation of anti-insulin receptor antibodies as potential novel therapies for human insulin receptoropathy using cell culture models.

Authors:  Gemma V Brierley; Kenneth Siddle; Robert K Semple
Journal:  Diabetologia       Date:  2018-04-27       Impact factor: 10.122

Review 4.  The Insulin Receptor: An Important Target for the Development of Novel Medicines and Pesticides.

Authors:  Xiaohong Zhang; Xuezhen Zhu; Xiaoyang Bi; Jiguang Huang; Lijuan Zhou
Journal:  Int J Mol Sci       Date:  2022-07-14       Impact factor: 6.208

5.  Anti-Insulin Receptor Antibodies Improve Hyperglycemia in a Mouse Model of Human Insulin Receptoropathy.

Authors:  Gemma V Brierley; Hannah Webber; Eerika Rasijeff; Sarah Grocott; Kenneth Siddle; Robert K Semple
Journal:  Diabetes       Date:  2020-08-17       Impact factor: 9.461

  5 in total

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