Literature DB >> 28597214

[Diagnosis of ischemia and revascularization in patients with ventricular tachyarrhythmia].

Thomas Deneke1, Carsten W Israel2.   

Abstract

Sustained ventricular tachyarrhythmia usually occurs on the basis of structural heart disease, particularly coronary heart disease (CAD). Although monomorphic ventricular tachycardia (VT) appears mainly in patients with CAD, it is typically not triggered by acute ischemia, in contrast to polymorphic VT or ventricular fibrillation (VF). To judge if VT is caused by acute ischemia is even more difficult in context with an elevated highly sensitive troponin T which is generally elevated in sustained VT because tachycardia in chronic stable coronary artery sclerosis causes a mismatch between increased oxygen demand and limited oxygen supply. Therefore, acute coronary angiography and revascularization may frequently not be necessary in monomorphic VT, will usually not improve rhythm stabilization, and may lead to misinterpretation of monomorphic VT being caused by a coronary stenosis. This can lead to withholding antiarrhythmic therapy after revascularization since it is assumed that the cause of VT has been treated. On the other hand, acute coronary angiography and revascularization are useful in polymorphic VT/VF, ECG signs of ischemia, or typical chest pain before occurrence of VT/VF. Coronary angiography should also be performed in patients with VT with newly diagnosed reduced left ventricular function and before catheter ablation.

Entities:  

Keywords:  Coronary angiography; Monomorphic; Polymorphic; Troponin; Ventricular fibrillation

Mesh:

Substances:

Year:  2017        PMID: 28597214     DOI: 10.1007/s00399-017-0515-1

Source DB:  PubMed          Journal:  Herzschrittmacherther Elektrophysiol        ISSN: 0938-7412


  10 in total

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  10 in total
  2 in total

Review 1.  Acute management of ventricular tachycardia.

Authors:  Thomas Deneke; Karin Nentwich; Elena Ene; Artur Berkovitz; Kai Sonne; Philipp Halbfaß
Journal:  Herzschrittmacherther Elektrophysiol       Date:  2020-02-06

2.  Complement C1q/Tumor Necrosis Factor-Related Protein-3 (CTRP3) is Significantly Decreased in Patients with Heart Failure and Closely Related with Ventricular Tachycardia.

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Journal:  Acta Cardiol Sin       Date:  2021-05       Impact factor: 2.672

  2 in total

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