Literature DB >> 28515064

Genetics of serum carotenoid concentrations and their correlation with obesity-related traits in Mexican American children.

Vidya S Farook1, Lavanya Reddivari2, Srinivas Mummidi1, Sobha Puppala3, Rector Arya1, Juan Carlos Lopez-Alvarenga1, Sharon P Fowler4, Geetha Chittoor5, Roy G Resendez1, Birunda Mohan Kumar6, Anthony G Comuzzie3, Joanne E Curran1, Donna M Lehman4, Christopher P Jenkinson1, Jane L Lynch7, Ralph A DeFronzo4, John Blangero1, Daniel E Hale7, Ravindranath Duggirala8, Jairam Kp Vanamala9,10.   

Abstract

Background: Dietary intake of phytonutrients present in fruits and vegetables, such as carotenoids, is associated with a lower risk of obesity and related traits, but the impact of genetic variation on these associations is poorly understood, especially in children.Objective: We estimated common genetic influences on serum carotenoid concentrations and obesity-related traits in Mexican American (MA) children.Design: Obesity-related data were obtained from 670 nondiabetic MA children, aged 6-17 y. Serum α- and β-carotenoid concentrations were measured in ∼570 (α-carotene in 565 and β-carotene in 572) of these children with the use of an ultraperformance liquid chromatography-photodiode array. We determined heritabilities for both carotenoids and examined their genetic relation with 10 obesity-related traits [body mass index (BMI), waist circumference (WC), high-density lipoprotein (HDL) cholesterol, triglycerides, fat mass (FM), systolic and diastolic blood pressure, fasting insulin and glucose, and homeostasis model assessment of insulin resistance] by using family data and a variance components approach. For these analyses, carotenoid values were inverse normalized, and all traits were adjusted for significant covariate effects of age and sex.
Results: Carotenoid concentrations were highly heritable and significant [α-carotene: heritability (h2) = 0.81, P = 6.7 × 10-11; β-carotene: h2 = 0.90, P = 3.5 × 10-15]. After adjusting for multiple comparisons, we found significant (P ≤ 0.05) negative phenotypic correlations between carotenoid concentrations and the following traits: BMI, WC, FM, and triglycerides (range: α-carotene = -0.19 to -0.12; β-carotene = -0.24 to -0.13) and positive correlations with HDL cholesterol (α-carotene = 0.17; β-carotene = 0.24). However, when the phenotypic correlations were partitioned into genetic and environmental correlations, we found marginally significant (P = 0.051) genetic correlations only between β-carotene and BMI (-0.27), WC (-0.30), and HDL cholesterol (0.31) after accounting for multiple comparisons. None of the environmental correlations were significant.Conclusions: The findings from this study suggest that the serum carotenoid concentrations were under strong additive genetic influences based on variance components analyses, and that the common genetic factors may influence β-carotene and obesity and lipid traits in MA children.
© 2017 American Society for Nutrition.

Entities:  

Keywords:  cardiometabolic traits; childhood obesity; common genetic influences; heritability; α-carotene; β-carotene

Mesh:

Substances:

Year:  2017        PMID: 28515064      PMCID: PMC5486195          DOI: 10.3945/ajcn.116.144006

Source DB:  PubMed          Journal:  Am J Clin Nutr        ISSN: 0002-9165            Impact factor:   7.045


  45 in total

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Review 5.  The Genetics of Pediatric Obesity.

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8.  Gene polymorphisms and gene scores linked to low serum carotenoid status and their associations with metabolic disturbance and depressive symptoms in African-American adults.

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6.  Serum carotenoids and Pediatric Metabolic Index predict insulin sensitivity in Mexican American children.

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