Literature DB >> 28460013

Delayed, oral pharmacological inhibition of calpains attenuates adverse post-infarction remodelling.

Marcos Poncelas1, Javier Inserte1,2, David Aluja1, Victor Hernando1, Ursula Vilardosa1, David Garcia-Dorado1,2.   

Abstract

Calpains activate during myocardial ischemia-reperfusion and contribute to reperfusion injury. Studies in transgenic animals with altered calpain/calpastatin system subjected to permanent ischemia suggest that calpains are also involved in post-infarction remodelling and heart failure. AIMS: To determine whether delayed oral administration of the calpain inhibitor SNJ-1945 reduces adverse myocardial remodelling and dysfunction following transient coronary occlusion. METHODS AND
RESULTS: Male Sprague-Dawley rats were subjected to 30 min of ischemia followed by 21 days of reperfusion and received the calpain inhibitor SNJ-1945 intraperitoneally at the onset of reperfusion (Acute group), orally starting after 24 h of reperfusion and for 14 days (Chronic group), or the combination of both treatments. Calpain-1 and calpain-2 protein content increased and correlated with higher calpain activity in control hearts. Administration of SNJ-1945 attenuated calpain activation, and reduced scar expansion, ventricular dilation and dysfunction in both acute and chronic groups. Acute treatment reduced infarct size in hearts reperfused for 24 h and inflammation measured after 3 days. Delayed, chronic oral administration of SNJ-1945 attenuated inflammation, cardiomyocyte hypertrophy and collagen infiltration in the non-infarcted myocardium at 21 days in correlation with increased levels of IĸB and reduced NF-ĸB activation. In cultured fibroblasts, SNJ-1945 attenuated TGF-β1-induced fibroblast activation.
CONCLUSIONS: Our data demonstrate for the first time that long-term calpain inhibition is possible with delayed oral treatment, attenuates adverse post-infarction remodelling, likely through prevention of NF-ĸB activation, and may be a promising therapeutic intervention to prevent adverse remodelling and heart failure in patients with acute myocardial infarction. Published on behalf of the European Society of Cardiology. All rights reserved.
© The Author 2017. For permissions please email: journals.permissions@oup.com.

Entities:  

Keywords:  Calpain; Ischemia/reperfusion; Post-infarction remodelling

Mesh:

Substances:

Year:  2017        PMID: 28460013     DOI: 10.1093/cvr/cvx073

Source DB:  PubMed          Journal:  Cardiovasc Res        ISSN: 0008-6363            Impact factor:   10.787


  18 in total

Review 1.  Calpains as Potential Therapeutic Targets for Myocardial Hypertrophy.

Authors:  David Aluja; Sara Delgado-Tomás; Marisol Ruiz-Meana; José A Barrabés; Javier Inserte
Journal:  Int J Mol Sci       Date:  2022-04-07       Impact factor: 6.208

2.  Over-expression of calpastatin attenuates myocardial injury following myocardial infarction by inhibiting endoplasmic reticulum stress.

Authors:  Shuai Li; Jian Ma; Jing-Bo Li; James C Lacefield; Douglas L Jones; Tian-Qing Peng; Meng Wei
Journal:  J Thorac Dis       Date:  2018-09       Impact factor: 2.895

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Journal:  Basic Res Cardiol       Date:  2019-03-15       Impact factor: 12.416

4.  Unraveling the Mechanisms by Which Calpain Inhibition Prevents Heart Failure Development.

Authors:  Xander H T Wehrens
Journal:  JACC Basic Transl Sci       Date:  2018-08-28

5.  Targeting Calpain for Heart Failure Therapy: Implications From Multiple Murine Models.

Authors:  Yihui Wang; Biyi Chen; Chun-Kai Huang; Ang Guo; Jennifer Wu; Xiaoming Zhang; Rong Chen; Cheng Chen; William Kutschke; Robert M Weiss; Ryan L Boudreau; Kenneth B Margulies; Jiang Hong; Long-Sheng Song
Journal:  JACC Basic Transl Sci       Date:  2018-08-28

6.  Connexin 43 Deficiency Is Associated with Reduced Myocardial Scar Size and Attenuated TGFβ1 Signaling after Transient Coronary Occlusion in Conditional Knock-Out Mice.

Authors:  Laura Valls-Lacalle; Marta Consegal; Marisol Ruiz-Meana; Begoña Benito; Javier Inserte; Ignasi Barba; Ignacio Ferreira-González; Antonio Rodríguez-Sinovas
Journal:  Biomolecules       Date:  2020-04-23

7.  CaMKII/calpain interaction mediates ischemia/reperfusion injury in isolated rat hearts.

Authors:  Hong-Ting Lu; Ren-Qian Feng; Jia-Kun Tang; Jing-Jun Zhou; Feng Gao; Jun Ren
Journal:  Cell Death Dis       Date:  2020-05-21       Impact factor: 8.469

8.  Targeted inhibition of calpain in mitochondria alleviates oxidative stress-induced myocardial injury.

Authors:  Dong Zheng; Ting Cao; Lu-Lu Zhang; Guo-Chang Fan; Jun Qiu; Tian-Qing Peng
Journal:  Acta Pharmacol Sin       Date:  2020-09-23       Impact factor: 7.169

9.  Opposite Effects of Moderate and Extreme Cx43 Deficiency in Conditional Cx43-Deficient Mice on Angiotensin II-Induced Cardiac Fibrosis.

Authors:  Laura Valls-Lacalle; Corall Negre-Pujol; Cristina Rodríguez; Saray Varona; Antoni Valera-Cañellas; Marta Consegal; Jose Martínez-González; Antonio Rodríguez-Sinovas
Journal:  Cells       Date:  2019-10-22       Impact factor: 6.600

10.  Inhibition of GPR35 Preserves Mitochondrial Function After Myocardial Infarction by Targeting Calpain 1/2.

Authors:  Ken Chen; Lei He; Yong Li; Xiuchuan Li; Chenming Qiu; Haifeng Pei; Dachun Yang
Journal:  J Cardiovasc Pharmacol       Date:  2020-06       Impact factor: 3.271

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