Literature DB >> 2845344

Extensive defects of mitochondrial electron-transfer chain in muscular cytochrome c oxidase deficiency.

M Tanaka1, S Miyabayashi, M Nishikimi, H Suzuki, Y Shimomura, K Ito, K Narisawa, K Tada, T Ozawa.   

Abstract

This study was undertaken to estimate the extent of molecular defects in the mitochondrial electron-transfer chain of a patient with mitochondrial myopathy. Biochemical and immunochemical studies were performed on the skeletal muscle mitochondria. Spectrophotometry and enzyme activity measurements localized a definite defect at the segment of cytochrome c oxidase (complex IV) of the electron-transfer chain. Immunoblotting and immunoprecipitation studies using the anti-complex IV antibody revealed that the contents of subunits 1, 4, 5, 6, and 7 of complex IV were markedly diminished and that subunit 2 was almost absent. Immunohistochemistry of the muscle tissue revealed a considerable accumulation of immunoreactive materials of complex IV in the ragged-red fibers. The immunoblots using the anti-NADH-ubiquinone oxidoreductase antibody demonstrated that the contents of NADH-ubiquinone oxidoreductase subunits were 47% of control and that the contents of three subunits were considerably decreased. The contents of ubiquinol-cytochrome c oxidoreductase subunits were also somewhat low (77% of control) and one of the minor contaminants detected in the control was completely absent. High-resolution one-dimensional sodium dodecyl sulfate-urea-gel electrophoresis disclosed that six additional unidentified polypeptides in the control were markedly diminished or completely missing. These results demonstrate that the molecular defects in the mitochondrial electron-transfer chain are more extensive than would be expected from either spectral analysis or enzyme activity measurements alone, and involve not only complex IV but also NADH-ubiquinone oxidoreductase and ubiquinol-cytochrome c oxidoreductase and several unidentified mitochondrial proteins.

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Year:  1988        PMID: 2845344     DOI: 10.1203/00006450-198810000-00006

Source DB:  PubMed          Journal:  Pediatr Res        ISSN: 0031-3998            Impact factor:   3.756


  5 in total

1.  Defective pattern of mitochondrial respiratory enzymes in mitochondrial myopathy.

Authors:  S Miyabayashi; K Haginoya; H Hanamizu; K Iinuma; K Narisawa; K Tada
Journal:  J Inherit Metab Dis       Date:  1989       Impact factor: 4.982

Review 2.  Organelle pathology in metabolic neuromuscular disease: an overview.

Authors:  L E Becker
Journal:  Can J Vet Res       Date:  1990-01       Impact factor: 1.310

Review 3.  Oxidative stress in mitochondria: decision to survival and death of neurons in neurodegenerative disorders.

Authors:  Makoto Naoi; Wakako Maruyama; Masayo Shamoto-Nagai; Hong Yi; Yukihiro Akao; Masashi Tanaka
Journal:  Mol Neurobiol       Date:  2005       Impact factor: 5.590

4.  Clinical and biochemical phenotype of the MELAS mutation.

Authors:  S Miyabayashi; H Hanamizu; R Nakamura; J I Hayashi; K Tada
Journal:  J Inherit Metab Dis       Date:  1993       Impact factor: 4.982

5.  Fatal infantile mitochondrial cardiomyopathy and myopathy with heterogeneous tissue expression of combined respiratory chain deficiencies.

Authors:  J Müller-Höcker; H Ibel; I Paetzke; T Deufel; W Endres; B Kadenbach; J M Gokel; G Hübner
Journal:  Virchows Arch A Pathol Anat Histopathol       Date:  1991
  5 in total

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