Literature DB >> 28403038

Lung carcinogenesis and fibrosis taken together: just coincidence?

Ioanna Giopanou1, Kristina A M Arendt, Georgios T Stathopoulos.   

Abstract

PURPOSE OF REVIEW: The pathogenesis of lung cancer and pulmonary fibrotic disorders partially overlaps. This review focuses on the common features of the two disease categories, aimed at advancing our translational understanding of their pathobiology and at fostering the development of new therapies. RECENT
FINDINGS: Both malignant and collagen-producing lung cells display enhanced cellular proliferation, increased resistance to apoptosis, a propensity for invading and distorting the lung parenchyma, as well as stemness potential. These characteristics are reinforced by the tissue microenvironment and inflammation seems to play an important adjuvant role in both types of disorders.
SUMMARY: Unraveling the thread of the common and distinct characteristics of lung fibrosis and cancer might contribute to a more comprehensive approach of the pathobiology of both diseases and to a pathfinder for novel and personalized therapeutic strategies.

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Mesh:

Year:  2017        PMID: 28403038      PMCID: PMC5641447          DOI: 10.1097/MCP.0000000000000390

Source DB:  PubMed          Journal:  Curr Opin Pulm Med        ISSN: 1070-5287            Impact factor:   3.155


  158 in total

1.  Survivin expression induced by endothelin-1 promotes myofibroblast resistance to apoptosis.

Authors:  Jeffrey C Horowitz; Iyabode O Ajayi; Priya Kulasekaran; David S Rogers; Joshua B White; Sarah K Townsend; Eric S White; Richard S Nho; Peter D R Higgins; Steven K Huang; Thomas H Sisson
Journal:  Int J Biochem Cell Biol       Date:  2011-10-25       Impact factor: 5.085

2.  TGF-beta driven lung fibrosis is macrophage dependent and blocked by Serum amyloid P.

Authors:  Lynne A Murray; Qingsheng Chen; Michael S Kramer; David P Hesson; Rochelle L Argentieri; Xueyang Peng; Mridu Gulati; Robert J Homer; Thomas Russell; Nico van Rooijen; Jack A Elias; Cory M Hogaboam; Erica L Herzog
Journal:  Int J Biochem Cell Biol       Date:  2010-10-29       Impact factor: 5.085

3.  Inhibition of mechanosensitive signaling in myofibroblasts ameliorates experimental pulmonary fibrosis.

Authors:  Yong Zhou; Xiangwei Huang; Louise Hecker; Deepali Kurundkar; Ashish Kurundkar; Hui Liu; Tong-Huan Jin; Leena Desai; Karen Bernard; Victor J Thannickal
Journal:  J Clin Invest       Date:  2013-02-22       Impact factor: 14.808

Review 4.  Does interstitial lung disease predispose to lung cancer?

Authors:  Craig E Daniels; James R Jett
Journal:  Curr Opin Pulm Med       Date:  2005-09       Impact factor: 3.155

Review 5.  Nintedanib: from discovery to the clinic.

Authors:  Gerald J Roth; Rudolf Binder; Florian Colbatzky; Claudia Dallinger; Rozsa Schlenker-Herceg; Frank Hilberg; Stefan-Lutz Wollin; Rolf Kaiser
Journal:  J Med Chem       Date:  2015-01-14       Impact factor: 7.446

6.  Endothelin-1 and transforming growth factor-beta1 independently induce fibroblast resistance to apoptosis via AKT activation.

Authors:  Priya Kulasekaran; Casey A Scavone; David S Rogers; Douglas A Arenberg; Victor J Thannickal; Jeffrey C Horowitz
Journal:  Am J Respir Cell Mol Biol       Date:  2009-02-02       Impact factor: 6.914

7.  Matrix stiffening and β1 integrin drive subtype-specific fibroblast accumulation in lung cancer.

Authors:  Marta Puig; Roberto Lugo; Marta Gabasa; Alícia Giménez; Adriana Velásquez; Roland Galgoczy; Josep Ramírez; Abel Gómez-Caro; Óscar Busnadiego; Fernando Rodríguez-Pascual; Pere Gascón; Noemí Reguart; Jordi Alcaraz
Journal:  Mol Cancer Res       Date:  2014-10-03       Impact factor: 5.852

8.  Inhibition of telomerase activity preferentially targets aldehyde dehydrogenase-positive cancer stem-like cells in lung cancer.

Authors:  Diego Serrano; Anne-Marie Bleau; Ignacio Fernandez-Garcia; Tamara Fernandez-Marcelo; Pilar Iniesta; Carlos Ortiz-de-Solorzano; Alfonso Calvo
Journal:  Mol Cancer       Date:  2011-08-09       Impact factor: 27.401

Review 9.  Mesothelial cells in tissue repair and fibrosis.

Authors:  Steven E Mutsaers; Kimberly Birnie; Sally Lansley; Sarah E Herrick; Chuan-Bian Lim; Cecilia M Prêle
Journal:  Front Pharmacol       Date:  2015-06-09       Impact factor: 5.810

10.  Histone modifications are responsible for decreased Fas expression and apoptosis resistance in fibrotic lung fibroblasts.

Authors:  S K Huang; A M Scruggs; J Donaghy; J C Horowitz; Z Zaslona; S Przybranowski; E S White; M Peters-Golden
Journal:  Cell Death Dis       Date:  2013-05-02       Impact factor: 8.469

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  1 in total

1.  miR-185 and miR-29a are similarly expressed in the bronchoalveolar lavage cells in IPF and lung cancer but common targets DNMT1 and COL1A1 show disease specific patterns.

Authors:  Eleni Bibaki; Eliza Tsitoura; Eirini Vasarmidi; George Margaritopoulos; Athina Trachalaki; Chara Koutoulaki; Theodora Georgopoulou; Demetrios A Spandidos; Nikos Tzanakis; Katerina M Antoniou
Journal:  Mol Med Rep       Date:  2018-03-19       Impact factor: 2.952

  1 in total

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