Literature DB >> 28387608

Common Polymorphisms in IFI16 and AIM2 Genes Are Associated With Periodontal Disease.

Julie T Marchesan1,2, Yizu Jiao1, Kevin Moss2, Kimon Divaris3, William Seaman4, Jennifer Webster-Cyriaque4,5, Shaoping Zhang1,2, Ning Yu6, Catharine Song1, Sompop Bencharit7, Ricardo Teles1,2, Steven Offenbacher1,2.   

Abstract

BACKGROUND: The single nucleotide polymorphism (SNP) context of a previously identified periodontitis-associated locus is investigated, and its association with microbial, biologic, and periodontal disease clinical parameters is examined.
METHODS: A 200-kb spanning region of 1q12 previously highlighted in a genome-wide association scan among 4,766 European American individuals (SNP rs1633266) was annotated. Two haplotype blocks were selected. Association of these polymorphisms with data on microbial plaque composition, gingival crevicular fluid (GCF)-interleukin (IL)-1β levels, and clinical parameters of periodontal disease were examined. Descriptive analysis of IFI16 and AIM2 protein expression in gingival tissues from healthy individuals (n = 2) and individuals with chronic periodontitis (n = 2) was done via immunohistochemistry.
RESULTS: The highlighted locus is a 100-kb region containing the interferon γ-inducible protein 16 (IFI16) and absent in melanoma 2 (AIM2) genes. Two haplotype blocks, rs6940 and rs1057028, were significantly associated with increased extent bleeding on probing and levels of microorganisms Porphyromonas gingivalis, Tannerella forsythia, and Campylobacter rectus (P ≤0.05). Haplotype block rs1057028 was also significantly associated with pathogens Fusobacterium nucleatum and Aggregatibacter actinomycetemcomitans, increased GCF-IL-1β levels, and extent of probing depth ≥4 mm (P ≤0.05). Prevalence of severe periodontitis (biofilm-gingival interface P3 classification) was positively associated with haplotype block rs1057028. Similar trends were observed for haplotype block rs1057028. IFI16 and AIM2 protein expression was observed in multiple cell types of gingival tissues, including inflammatory cells.
CONCLUSION: This study found IFI16 and AIM2 SNPs associated with higher levels of periodontal microorganisms and an increased percentage of periodontal disease clinical parameters, suggesting the need for functional studies and additional fine-mapping of variants in the 1q12-locus.

Entities:  

Keywords:  Genetics; immunity, innate; periodontitis; polymorphism, genetic

Mesh:

Substances:

Year:  2017        PMID: 28387608      PMCID: PMC5695043          DOI: 10.1902/jop.2017.160553

Source DB:  PubMed          Journal:  J Periodontol        ISSN: 0022-3492            Impact factor:   6.993


  46 in total

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3.  Periodontal findings in adult twins.

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9.  A genome-wide association study of periodontitis in a Japanese population.

Authors:  S Shimizu; Y Momozawa; A Takahashi; T Nagasawa; K Ashikawa; Y Terada; Y Izumi; H Kobayashi; M Tsuji; M Kubo; Y Furuichi
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10.  SNPnexus: a web server for functional annotation of novel and publicly known genetic variants (2012 update).

Authors:  Abu Z Dayem Ullah; Nicholas R Lemoine; Claude Chelala
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3.  In Defense of Flossing: Part II-Can We Agree It's Premature to Claim Flossing Is Ineffective to Help Prevent Periodontal Diseases?

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Review 4.  AIM2 Inflammasome's First Decade of Discovery: Focus on Oral Diseases.

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Review 5.  Role of inflammasomes in the pathogenesis of periodontal disease and therapeutics.

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7.  Impaired function of epithelial plakophilin-2 is associated with periodontal disease.

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Review 9.  Inflammasomes in Alveolar Bone Loss.

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Journal:  Front Immunol       Date:  2021-06-09       Impact factor: 7.561

10.  Inflammasomes as contributors to periodontal disease.

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Journal:  J Periodontol       Date:  2020-08-06       Impact factor: 6.993

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