Literature DB >> 28371683

Association between serum alkaline phosphatase and coronary artery calcification in a sample of primary cardiovascular prevention patients.

Loïc Panh1, Jean Bernard Ruidavets2, Hervé Rousseau3, Antoine Petermann3, Vanina Bongard2, Emilie Bérard2, Dorota Taraszkiewicz1, Olivier Lairez1, Michel Galinier1, Didier Carrié1, Jean Ferrières4.   

Abstract

BACKGROUND AND AIMS: A high level of serum alkaline phosphatase (ALP) is associated with an increased risk of mortality and myocardial infarction. ALP hydrolyses inorganic pyrophosphate, which is a strong inhibitor of calcium phosphate deposition. The aim of this study was to determine whether ALP is associated with the coronary artery calcium score (CACS).
METHODS: We examined the association of CACS, assessed by computed tomography scanning, and ALP, in 500 patients consecutively recruited, free of cardiovascular disease. The CACS were categorized into two groups: no calcification (CACS = 0) (n = 187) and with calcification (CACS>0) (n = 313). ALP activity was divided into three tertile groups: low ALP level (<55 IU/L), intermediate (55-66 IU/L) and high ALP level (>66 IU/L).
RESULTS: The mean age was 60.9 ± 10.8 years, 49.6% of the patients were women. ALP ranged from 22 to 164 IU/L (mean 62.6 IU/L, SD 19.3). In univariate analysis, traditional cardiovascular risk factors, statin use (p = 0.001), and ALP (p = 0.001) were significantly associated with CACS. After adjusting for cardiovascular risk factors, only age (p = 0.001) and sex (p = 0.001) were independently associated with CACS. Compared to the tertile group with low levels of ALP, the intermediate tertile group [OR 2.11, 95% CI (1.12; 3.96), p = 0.02], as well as the high tertile group [OR 3.89, 95% CI (2.01; 7.54), p = 0.001)], was independently associated with CACS.
CONCLUSIONS: In patients free of cardiovascular disease, high ALP levels are positively and independently associated with coronary artery calcification. The metabolic pathway of ALP and inorganic pyrophosphate could be a target for new therapies against vascular calcification.
Copyright © 2017 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Alkaline phosphatase; Calcification; Coronary artery disease; Statin

Mesh:

Substances:

Year:  2017        PMID: 28371683     DOI: 10.1016/j.atherosclerosis.2017.03.030

Source DB:  PubMed          Journal:  Atherosclerosis        ISSN: 0021-9150            Impact factor:   5.162


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